Recent studies ofretinal damage due to ultrashort laser pulses' have shown interesting behavior. Laser induced retinal damage for ultrashort (i.e. less than ins) laser pulses is produced at lower energies than in the nanosecond to microsecond laser pulse regime and the energy required for hemorrhagic lesions is much greater times greater than for the nanosecond regime. We investigated the tissue effects exhibited in histopathology ofretinal tissues exposed to ultrashort laser pulses.