Relative and Combined Effects of Ethanol and Protein Deficiency on Zinc, Iron, Copper, and Manganese Contents in Different Organs and Urinary and Fecal Excretion

Polyenylphosphatidylcholine Protects Against Alcohol but Not Iron‐Induced Oxidative Stress in the Liver

“…We confirmed that alcohol feeding leads to a decline of hepatic nonherne iron (Olynyk et al, 1995;Rouach et al, 1997) that could be related to the increased urinary excretion of iron found in ethanol-fed rats (Gonzalez-Reimers et al, 1998) or to other disturbances of iron metabolism (Houglum, 1996;Pietrangelo, 1996;Rouach et al, 1997).…”

Tissue Mineral Distributions are Differentially Modified by Dietary Protein Deficiency and a Murine Nematode Infection

“…In the absence of infection, the impact of protein deficiency on body mineral distribution has been studied almost exclusively in the liver, where the findings are surprisingly inconsistent. Reduced liver zinc concentrations were found in most animal studies [14][15][16], except that of Kilicalp et al [17] who showed no change. Whereas some authors have reported lower copper [15,16] concentrations in livers of protein-deficient rats, others have demonstrated no change of copper, manganese [17,18], calcium, and magnesium concentrations [17].…”

Fluorescence signaling of Zn2+ levels in synthetic urine by dipicolylamine-armed hydroxynaphthalimide

“…The levels of zinc in healthy adults are approximately 1 g/mL in serum and 0.5 g/mL in urine [6]. The urine Zn level is known to be related to some important biological, habitual, and environmental factors [7][8][9]. Thus, determination of zinc metal ions in urine can be used as a diagnostic tool in the clinical test of metabolic diseases and for assessing the effects of industrial, occupational, and environmental exposures [10].…”