Relationship Between Primary Acquired Nasolacrimal Duct Obstruction, Paranasal Abnormalities and Nasal Septal Deviation

Abstract

Objectives: This study aimed to investigate the relationship between nasal septal deviation and paranasal abnormalities in the etiology of primary acquired nasolacrimal duct obstruction (PANDO). Methods: A total of 37 (8 men, 29 women) patients (48 eyes with PANDO) between the ages 20 and 77 years (mean age, 52.8 ± 13.1 years) were included in the study. In the axial sections of paranasal sinus computed tomography, the transverse diameters at the most upper part and the most distal part of the nasolacrimal canal were separately measured. In addition, the relationships between PANDO and nasal septal deviation, inferior turbinate hypertrophy, agger nasi cells, paradoxical middle turbinate, concha bullosa, and the angle between the bony inferior turbinate and medial wall of the maxillary sinus were investigated. Results: A statistically significant relationship was found between PANDO and the axial location of septal deviation classification, axial angle of septal deviation classification, paradoxical middle turbinate, angle between the bony inferior turbinate and medial wall of the maxillary sinus and inferior meatus measure. Conclusion: The authors concluded that the location and angle of the nasal septal deviation in the axial plane, width of the angle between the bony inferior turbinate and medial wall of the maxillary sinus, inferior meatus measure, and paradoxical middle turbinate may be effective factors in the etiology of PANDO.

Anatomic characteristics of primary acquired nasolacrimal duct obstruction: a comparative computed tomography study

“…While it is still uncertain how the narrower inferior turbinate angle is related to the obstruction, this may be because the narrower inferior turbinate angle affects the opening site of the lacrimal drainage system. In contrast to some previous results (15,(17)(18)(19)(20), nasal septal deviation, concha bullosa, and inferior turbinate thickness were found to be not associated with PANDO. The prevalence of sinus inflammation shown by CT in the healthy controls was similar to the result of a previous study (23).…”

“…This study applied widely used methods to measure the nasal quantitative structure. Two nasal structure parameters were measured on the coronary section according to previously reported methods ( 20 , 21 ), including inferior turbinate thickness and inferior turbinate angle (the angle between the bony inferior turbinate and the medial wall of the maxillary sinus), which reflected the relative position of the inferior turbinate ( Figure 1F ). Nasal abnormalities included nasal septal deviation, concha bullosa, and the inflammation of the frontal sinus, maxillary sinus, ethmoid sinus, osteomeatal complex (the common channel linking the frontal sinus, anterior ethmoid air cells, and the maxillary sinus to the middle meatus), and agger nasi cells (the most anterior ethmoidal air cells lying inferior to the frontal recess).…”

Rhinological Status of Patients with Nasolacrimal Duct Obstruction

“…Besides, in the present study, we analyzed the distribution of the volume of the inferior nasal conchae in patients of different groups, since the literature contains information that hypertrophy of the inferior nasal conchae may be one of the factors contributing to the development of nasolacrimal duct obstruction. 11 12 13 Fig. 3b shows the process of measuring the volume of the inferior nasal concha.…”

Incidences Of Nasal Septal Deviation And Its Effect On Surgical Success In Patients With Congenital Nasolacrimal Dacryostenosis

“…5 The bony structures of the nasolacrimal duct are located in the lateral nasal wall, suggesting that the structural abnormalities of this duct system may be affected by pathologies of the nasal cavity. 6 In a previous study on adult patients, a statistically signi cant relationship was found between acquired nasolacrimal duct obstruction (acquired NLDO) and NSD. 7 There are other studies that examined nasal pathologies and nasolacrimal duct obstruction in adult patients.…”