Rehabilitation of a patient with non-syndromic partial oligodontia

Abstract

Oligodontia is defined as a congenital tooth agenesis with the absence of six or more permanent teeth. This clinical report describes a patient with non-syndromic partial oligodontia, with retained deciduous teeth and the absence of 16 permanent teeth. Anterior esthetic problems were caused by interarch tooth size discrepancy, interdental space, aberrant tooth dimensions, and the absence of centric contacts of the anterior teeth. Prosthetic restoration after orthodontic and implant treatment was performed with a multi-disciplinary team approach. Favorable functional and esthetic results were obtained using a definitive prosthesis.

Non-syndromic oligodontia: A case report

“…The relationship between dental agenesis and the presence of dental or skeletal malocclusions has been described by various authors. The results coincide that depending on the ethnic group, the upper incisors can present pro-or retro inclination, the maxillae can have a reduced size and present a reduction of the vertical dimension being unfavorable both aesthetically and functionally [2] [7].…”

“…Syndromic oligodontia can occur as a dental trait attributed to mutations in genes: PAX9, MSX1, AXIN2, EDA and EDAR or concomitantly with systemic diseases such as ectodermal dysplasia, Down syndrome, Nance-Horan syndrome, Rieger syndrome, Seckel syndrome, Wolf-Hirschhorn syndrome, Klippel-Feil syndrome, Van der Woude syndrome and cleft lip and palate, among others [2,3,4]. Non-syndromic oligodontia could be due to environmental causes, medications, and nutritional imbalances as isolated factors.…”

Suspected Non-Syndromic Oligodontia – A Rare Case with 13 Agenesis

“…Orthodontic treatment can improve the spatial redistribution of the missing area, rearrangement of teeth, and correction of the tooth axis. 7 The best time for orthodontic treatment of patients with agenesis of mandibular second premolars is usually early adolescence. This is when most of the remaining developing permanent teeth are erupting, and most of the facial growth has happened.…”

“…Many pieces of literature stated that incidence of oligodontia relatively low and could vary from 0.08% to 0.16% [3][4][5] with a higher frequency has been noted in females than males with no difference in the distribution of missing teeth over maxilla/ mandible and left and right sides. 6 Oligodontia can occur in association with various genetic syndromes such as ectodermal dysplasia, Van Der Woude syndrome, Nance-Horan Syndrome, Down syndrome, Rieger syndrome, and cleft lip and palate 7 or as a non-syndromic isolated familial trait, or as an infrequent finding. 8 Among the syndromic form of oligodontia, ectodermal dysplasia is the most common form.…”

Implant Therapy of Non-Syndromic Severe Oligodontia: A Clinical Report

“…Oligodontia is a number anomaly and is the most common dental anomaly that can affect patients with syndrome or non-syndrome [13]; Some studies reported mutations in the genes that were associated with nonsyndromic oligodontia, mutation of gene MXS1 or PAX9 may create an impairment of one or more molecular processes that regulate tooth formation [1,14] and some studies report that it may be greater in women than in men [3].…”