“…At variance, significant changes of the ulnar sensory nerve action potential and MNCV were found in the unaffected side of stroke patients, suggesting an increased risk of NCS abnormalities due to risk factors for polyneuropathy, such as diabetes mellitus (Paoloni et al, 2010). In our view, although none of the NCS measures recorded in this study in the two upper limbs fell outside the normal range values (Puksa et al, 2003;Qrimli et al, 2016;Alber, 2017), it is plausible that all the above-mentioned mechanisms, and in particular the "subclinical" involvement of the lower motor neuron in UMNS (Hara et al, 2000), might have contributed to the NCS asymmetries here detected. Furthermore, we also argue that spasticity might not have played a crucial role in the NCS differences observed between the upper limbs, given that we detected only a significant inverse correlation between wrist flexor MAS score and ulnar nerve ADM CMAP, and a significant direct correlation between finger flexor MAS score and ulnar nerve ADM DML.…”