“…Several patterns of injury such as focal nodular hyperplasia, hepatocellular adenoma [ 27 ], hepatocellular carcinoma [ 28 ], peliosis hepatis [ 29 , 30 ], spontaneous hepatic rupture [ 31 ] and, specially, cholestasis hepatitis [ 11 , 24 , 25 , 26 , 32 , 33 , 34 , 35 , 36 ], have been described. However, in a recent study, AAS hepatotoxicity was associated with a distinct phenotype, characterized by considerable total bilirubin (TB) elevations independent of type of damage, in addition to low values of transaminases and alkaline phosphatase, compared to values in liver injury due to convencional drugs and herbs [ 10 , 11 , 24 , 25 , 26 , 37 ].…”