“…Potential mechanisms for the development of osteonecrosis may be partially dependent on the type of treatment but include kinking of periosteal blood vessels resulting from torsional deformity, synovitis associated with the trauma, vascular tamponade resulting from intracapsular hematoma, timing of treatment, and location of fixation devices within the epiphysis. Multiple treatment algorithms that were developed to address these theoretical causes include intentional delay in treatment [11], preoperative traction [13,19,20], both open and closed urgent reduction and fixation [8,17], and open or percutaneous capsulotomy [8,12,19] and are all associated with developing osteonecrosis. Patients with unstable SCFE often presented with substantially more deformity, often correctable, and were more likely to undergo attempted reduction, either intentional or inadvertent.…”