Radicular (Type I) dentin dysplasia

Perl, Thomas R.; Farman, Allan G.

doi:10.1016/0030-4220(77)90060-3

Cited by 14 publications

(5 citation statements)

References 9 publications

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“…DD is usually an autosomal dominant condition [1], but in this patient, there was no familial history of the disease, so he is considered to be a first generation sufferer. Teeth with radiographic or histologic features of DD occur in a number of disorders such as calcinosis, Ehlers-Danlos syndrome, and the brachioskeletogenital syndrome [9]. Some association has also been reported between dentine dysplasia and osseous changes in addition to sclerotic bone formation [10] but our patient had no signs of other pathologic conditions.…”

Section: Discussionmentioning

confidence: 80%

Dentin dysplasia type I: a case report and review of the literature

et al. 2010

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IntroductionDentin dysplasia is a rare hereditary disturbance of dentin formation characterized by defective dentin development with clinically normal appearing crowns, severe hypermobility of teeth and spontaneous dental abscesses or cysts. Radiographic analysis shows obliteration of all pulp chambers, short, blunted and malformed or absent roots and peri-apical radiolucencies of non carious teeth.Case presentationWe present a case of dentin dysplasia type I in a 12-year-old Iranian boy, and the clinical, radiographic and histopathologic findings of this condition and treatment are described.ConclusionsThere are still many inconclusive issues in the diagnosis and management of patients with dentin dysplasia. The diagnostic features of this rare disturbance will remain incompletely defined until additional cases have been described. Early diagnosis of the condition and initiation of effective regular dental treatments may help these patients to prevent or delay loss of dentition.

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Section: Discussionmentioning

confidence: 80%

Dentin dysplasia type I: a case report and review of the literature

et al. 2010

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“…DD is usually an autosomal dominant condition, and in two of our patients, there was a familial history of the disease, and the patient with no family history is considered to be a first generation sufferer. Teeth with radiographic or histological features of DD occur in a number of disorders such as Ehlers–Danlos syndrome and the brachioskeletogenital syndrome 11. There are reports that have suggested possible variations in the morphology of teeth affected by this type of dysplasia which was seen in our patients 12 13.…”

Section: Discussionmentioning

confidence: 54%

Dentin dysplasia type I

et al. 2013

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Dentin dysplasia type I is a rare hereditary disturbance of dentin formation characterised clinically by nearly normal appearing crowns and hypermobility of teeth that affects one in every 100,000 individuals and manifests in both primary and permanent dentitions. Radiographic analysis shows obliteration of all pulp chambers, short, blunted, and malformed roots, and periapical radiolucencies of non-carious teeth. This paper presents three cases demonstrating classic features of type I dentin dysplasia.

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“…Another aspect which makes root canal location more difficult is dentin morphology. In patients with DD-1, the teeth have softer dentin that lacks a regular tubular morphology and may contain irregular channels [ 44 ]. Thus, regardless of the location of the root canal, files with cutting tips, which are frequently used to scout root canals and penetrate calcifications, might also penetrate the soft dentin and increase the risk of perforation.…”

Section: Discussionmentioning

confidence: 99%

Guided endodontic treatment of multiple teeth with dentin dysplasia: a case report

et al. 2020

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Background To report the outcome of guided endodontic treatment (GET) of a case of dentin dysplasia with pulp canal calcification (PCC) and apical periodontitis based on the use of a 3D-printed template designed by merging cone-beam computed tomography (CBCT) and surface scan data. Case presentation A 12-year old female with radicular dentin dysplasia type I (DD-1) presented for endodontic treatment. Radiography revealed PCC in all teeth and apical radiolucency in seven teeth (12, 15, 26, 31, 32, 36 and 46). Tooth 36 had the most acute symptoms and was thus treated first by conventional access cavity preparation and root canal detection. Despite meticulous technique, the distal and mesiolingual canals were perforated. The perforations were immediately repaired with mineral trioxide aggregate, and the decision was made to switch to guided endodontic treatment for the remaining 6 teeth. CBCT and intraoral surface scans were acquired and matched using coDiagnostix planning software (Dental Wings Inc.), the respective drill positions for root canal location were determined, and templates were virtually designed and 3D-printed. The template was positioned on the respective tooth, and a customized drill was used to penetrate the calcified part of the root canal and perform minimally invasive access cavity preparation up to the apical region. All root canals were rapidly and successfully located with the templates. At 1-year follow-up, clear signs of apical healing were present in all treated teeth. Conclusions In patients with dentin dysplasia, conventional endodontic therapy is challenging. GET considerably facilitates the root canal treatment of teeth affected by dentin dysplasia.

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Radicular (Type I) dentin dysplasia

Cited by 14 publications

References 9 publications

Dentin dysplasia type I: a case report and review of the literature

Dentin dysplasia type I: a case report and review of the literature

Dentin dysplasia type I

Guided endodontic treatment of multiple teeth with dentin dysplasia: a case report

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