Pulmonary Manifestations in the Diffuse Collagen Diseases

Ellman, Philip; Cudkowicz, Leon

doi:10.1136/thx.9.1.46

Cited by 76 publications

(7 citation statements)

References 32 publications

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“…Further more, patients with IPF are reported to suffer simultaneously from other autoim mune diseases such as rheumatic arthritis [5,21], Sjogren's syndrome [2], sclero derma [20], and Hashimoto's disease [24]. In spite of these findings, thus far no spe cific autoantibodies against the lung cell surface prompting IPF have been identi fied.…”

Section: Introductionmentioning

confidence: 90%

Detection of Antibodies in Serum of Patients with Idiopathic Pulmonary Fibrosis against Isolated Rat Alveolar Type II Cells

Baumgartner

Schölmerich²,

Becker³

et al. 1987

Respiration

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The serum of 2 patients suffering from idiopathic pulmonary fibrosis (IPF) repeatedly demonstrated a positive antibody reaction against rat alveolar type II cells isolated by the use of a combination of elastase and trypsin for disaggregation and a Percoll density gradient for purification. Neither of the other 8 IPF patients examined nor any of the controls showed a positive reaction. Because the 2 patients with serum antibodies against alveolar type II cells did not show any antinuclear antibodies, it is concluded that the antibodies against the surface of rat alveolar type II cells found in the serum of IPF patients may be autoantibodies involved in the pathogenesis of IPF

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Section: Introductionmentioning

confidence: 90%

Detection of Antibodies in Serum of Patients with Idiopathic Pulmonary Fibrosis against Isolated Rat Alveolar Type II Cells

Baumgartner

Schölmerich²,

Becker³

et al. 1987

Respiration

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“…[1][2][3][4] Standard textbooks of pulmonary medicine relate the progressive dyspnea, small lung volume with elevated hemi diaphragms to ''an unspecified restriction in chest wall expansion'' generally ascribed to diaphragmatic weakness. 5 Investigators have hypothesized or measured defects in lung recoil, 1 phrenic nerve function 6 and diaphragmatic strength.…”

Section: Introductionmentioning

confidence: 99%

Pleural fibrosis mediates shrinking lungs syndrome in children

Krych

Fischer

Wylam

2008

Pediatric Pulmonology

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Summary. The etiology of restrictive lung physiology in SLS is not well-defined, and has been hypothesized to be due to defects in lung recoil, phrenic nerve function and diaphragmatic strength. We present a case of SLS in an adolescent in whom imaging and electrophysiology studies demonstrate pleural fibrosis as the fundamental defect accounting for the restrictive lung physiology. Pediatr Pulmonol. 2009; 44:90-92. ß

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“…Opie (1955). We have found in the literature eleven clinically described cases of systemic sclerosis with pulmonary hypertension verified by heart catheterization: Table IV, overleaf (Spain and Thomas, 1950;Austrian, McClement, Renzetti, Donald, Riley, and Cournand, 1951;Foss, 1954;Ellman and Cudkowicz, 1954;Bauer, 1955;Opie, 1955;Fischer and Hansen, 1956;Castleman and Kibbee, 1960;Soulie, Acar, Joly, Carlotti, Bernard, Azhrad, and Casillon du Perron, 1961;Pace, Decker, and Martin, 1963;Tourniaire, Blum, Tarlulier, and Deyrieux, 1964). As with the morbid anatomical and physiological studies, three groups may be discerned:…”

Section: Review Of the Literature A Morbid Anatomical Investigationsmentioning

confidence: 98%