Psychiatric Disorders in Patients With Fibromyalgia

Epstein, Steven A.; Kay, Gary G.; Clauw, Daniel J.; Heaton, Robert K.; Klein, Daniel N.; Krupp, Lauren; Kuck, Julie; Leslie, Vinita; Masur, David; Wagner, Mark T.; Waid, Randy; Zisook, Sidney

doi:10.1016/s0033-3182(99)71272-7

Cited by 337 publications

(207 citation statements)

References 26 publications

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“…para este estudio, el valor de la subescala de ansiedad-estado fue α=.93. De esta subescala, se tomó el percentil 75 (puntuación 31 en mujeres) como criterio clínicamente significativo de ansiedad elevada (Echeburúa, 2004). además, en función de la media y desviación típica (DT) obtenida en nuestra muestra (32.76±11.27) se calculó el punto de corte para el intervalo de +1Dt, coincidiendo con el percentil 90 (puntuación 44 en mujeres) y valores extremos de ansiedad.…”

Section: Variables E Instrumentosunclassified

La Escala de Ansiedad y Depresión Hospitalaria (HAD) en fibromialgia: Análisis de sensibilidad y especificidad

et al. 2015

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Section: Variables E Instrumentosunclassified

La Escala de Ansiedad y Depresión Hospitalaria (HAD) en fibromialgia: Análisis de sensibilidad y especificidad

et al. 2015

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“…52 Similarly, fibromyalgia syndrome, which overlaps with CFS, has been associated with major depression and dysthymia. 53,54 Interestingly, fibromyalgia syndrome has been associated with adverse childhood experiences. Moreover, specific personality traits which may be related to such adverse experiences (eg, unstable self-esteem), may also be associated with fibromyalgia, just as they are associated with dysthymia.…”

Section: Symptom and Illness Comorbiditymentioning

confidence: 99%

“…It will be recalled that dysthymia likewise is associated with a large number of comorbid conditions, such as neurodegenerative disorders, 1,61 traumatic brain injury, 62 and illnesses potentially involving viral components, such as fibromyalgia syndrome and chronic fatigue syndrome. 53,54 As such, the possibility ought to be considered that such coexisting illnesses are not simply correlates of the mood disorder, but may actually act to either precipitate or aggravate dysthymia.…”

Section: Cytokines and Depressive Illnessmentioning

confidence: 99%

Dysthymia: a review of pharmacological and behavioral factors

et al. 2000

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Although dysthymia, a chronic, low-grade form of depression, has a morbidity rate as high as that of major depression, and increases the risk for major depressive disorder, limited information is available concerning the etiology of this illness. In the present report we review literature concerning the biological and characterological features of dysthymia, the effectiveness of antidepressant treatments, the influence of stressors in the precipitation and maintenance of the disorder, and both quality of life and psychosocial correlates of the illness. We also provisionally suggest that dysthymia may stem from disturbances of neuroendocrine and neurotransmitter functioning (eg, corticotropin releasing hormone and arginine vasopressin within the hypothalamus, or alternatively monoamine variations within several extrahypothalamic sites), and may also involve cytokine activation. The central disturbances may reflect phenotypic variations of neuroendocrine processes or sensitization of such mechanisms. It is suggested that chronic stressor experiences or stressors encountered early in life lead to the phenotypic neurochemical alterations, which then favor the development of the dysthymic state. Owing to the persistence of the neurochemical disturbances, vulnerability to double depression is increased, and in this instance treatment with antidepressants may attenuate the symptoms of major depression but not those of the basal dysthymic state. Moreover, the residual features of depression following treatment may be indicative of underlying neurochemical disturbances, and may also serve to increase the probability of illness recurrence or relapse. Molecular Psychiatry (2000) 5, 242-261.

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“…In reality, though, many medical conditions arise where severe pain is reported but no specific pathology can be identified. These include conditions like chronic widespread pain and fibromyalgia, temporomandibular disorders, and chronic back pain (Aaron, Burke, & Buchwald, 2000;Epstein et al, 1999;Gatchel, 2002;Gatchel, Stowell, Wildenstein, Riggs, & Ellis, 2006;Gracely, Petzke, Wolf, & Clauw, 2002;Greene, 2001;Gremillion, 2000;Kight, Gatchel, & Wesley, 1999). When no pathology could be reliably identified, the disorder was termed psychogenic pain (i.e., the pain has a psychological basis).…”

Section: Theories Of Pain Biomedical Reductionismmentioning

confidence: 99%