“…Some patients experiencing severe COVID-19, the disease caused by the SARS-CoV-2 beta coronavirus, develop what is sometimes described as a “cytokine storm” or “cytokine release syndrome” characterized by the overstimulation of macrophages, dendritic cells and monocytes producing the cytokines interleukin 1 (IL1), interleukin 6 (IL6), interleukin 10 (IL10), tumor necrosis factor alpha (TNF-α), tumor necrosis factor beta (TNF-β) and ferritin [ 1 , 2 , 3 , 4 , 5 , 6 ]. These cytokines produce eosinopenia and lymphocytopenia characterized by low counts of eosinophils, CD8+ T cells, natural killer (NK) and naïve T-helper cells, simultaneously inducing naive B-cell activation, increased T-helper cell 17 (Th17) lymphocyte differentiation and the stimulation of monocyte and neutrophil recruitment [ 1 , 2 , 3 , 4 , 5 , 6 ]. Oddly, however, the release of cytokines interferon alpha (IFNα) and interferon gamma (IFNγ) are severely impaired in severe COVID-19 [ 1 , 2 , 3 , 4 , 5 , 6 ].…”