“…Analyses in current (e.g., Bertram et al, ; Eversole, Pappas, & Graham, ; Takayama, Miura, Yuasa, Kobayashi, & Hosoi, ) and ancient (e.g., Hinton, ; Hinton & Carlson, ; Hodges, ; Liryo da Silva, ; Richards, ; Richards & Brown, ; Sheridan, Mittler, Van Gerven, & Covert, ; Whittaker, Davies, & Brown, ) samples proposed that TMJ‐OA is probably associated with dental pathologies as caries and functional overload produced by malocclusal problems derived from dental attrition (DA) and antemortem tooth loss (AMTL) of the posterior dentition. Moreover, some evidence suggested that TMJ‐OA could be also secondary to bruxism, trauma, developmental abnormalities, the morphology of the skull and mandible, and even emotional stress (Bansal, ; Granados, ; Katsavrias, ; Rando & Waldron, ; Richards & Brown, ; Su, Liu, Yang, Shen, & Wang, ; Takayama et al, ; Wang et al, ; Widmalm et al, ).…”