Presumed Alcohol-Induced Ventricular Tachycardia Storm: A Case Report

Lima, Gian; Cardoso, Eduardo; Fiscus, Garret

doi:10.7759/cureus.8097

Cited by 1 publication

(2 citation statements)

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“…A relationship was demonstrated between chronic alcohol consumption and alcoholic cardiomyopathy, which is responsible for cardiac arrhythmias. On the other hand, there are many studies in the literature indicating a relationship between chronic alcohol consumption and an increased risk of AF in seemingly healthy individuals with no comorbidities (15) . It should be noted that the number of diagnosed HHS cases may not reflect the actual number of cases.…”

Section: Discussionmentioning

confidence: 99%

“…Alcohol can increase the release of catecholamines secreted by the adrenal medulla or secreted directly by the myocardium itself (12) . An increase in both systemic and intramyocardial catecholamine levels can result in the prolongation of the P wave, which may be associated with atrial arrhythmias (7,15) . However, Mäki et al did not observe any significant increase in catecholamine levels following alcohol consumption in individuals with episodes of AF.…”

Section: Pathophysiologymentioning

confidence: 99%

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Holiday heart syndrome: influence of alcohol on heart rhythm

Kostrzewska¹,

Całkosiński²,

Majewski³

et al. 2021

Pediatr Med Rodz

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The impact of alcohol on human health is widely known. There is a large body of research about positive (the “French paradox”) and negative effects of alcohol consumption. The relationship between excessive alcohol consumption and cardiac arrhythmias was first described in the 1970s in people who consumed alcohol heavily, mainly on weekends or holidays, but also in those who drank little or did not consume any alcohol. The term “holiday heart syndrome” was used for the first time by Philip Ettinger with reference to healthy people without cardiovascular disease. Excessive alcohol consumption can cause arrhythmias, most often in the form of atrial fibrillation. Ethanol and its metabolites have a toxic impact on cardiac myocytes; moreover, alcoholic cardiomyopathy accounts for one-third of all cases of non‐ischaemic dilated cardiomyopathy. It is possible to restore normal heart function through early diagnosis and cessation of alcohol consumption. The prevalence of holiday heart syndrome depends on the drinking habits of the study population. Holiday heart syndrome should be considered especially in patients without overt heart disease with a new onset of atrial fibrillation. Although relapses do occur, the clinical course is mild and specific antiarrhythmic therapy is usually not warranted. People diagnosed with cardiovascular disease benefit from minimising the amount of alcohol consumption. There is no safe amount of alcohol to drink and especially those with alcoholic cardiomyopathy should strive for abstinence in order to optimise treatment.

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Section: Discussionmentioning

confidence: 99%

Section: Pathophysiologymentioning

confidence: 99%