2004
DOI: 10.3949/ccjm.71.4.303
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Premenstrual dysphoric disorder: a review for the treating practitioner.

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Cited by 29 publications
(20 citation statements)
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“…The pathophysiology of PMS remains unknown, complex and multifactorial and yet to be fully clarified and may include the effect of progesterone on neurotransmitters such as serotonin, opioids, catecholamine and GABA, increased prolactin level or increased sensitivity to the effect of prolactin, insulin resistance, sensitivity to endogenous hormones, abnormal hypothalamic-pituitary-adrenal axis function, nutritional deficiencies, alteration of glucose metabolism, and fluid and electrolyte imbalance [13,16-18]. …”
Section: Introductionmentioning
confidence: 99%
“…The pathophysiology of PMS remains unknown, complex and multifactorial and yet to be fully clarified and may include the effect of progesterone on neurotransmitters such as serotonin, opioids, catecholamine and GABA, increased prolactin level or increased sensitivity to the effect of prolactin, insulin resistance, sensitivity to endogenous hormones, abnormal hypothalamic-pituitary-adrenal axis function, nutritional deficiencies, alteration of glucose metabolism, and fluid and electrolyte imbalance [13,16-18]. …”
Section: Introductionmentioning
confidence: 99%
“…It has not been identified fully so far and may be due to the effect of progesterone on neurotransmitters such as serotonin, opioids, catecholamines, or GABA. It may also be due to increased prolactin levels, increased sensitivity to the effects of prolactin, insulin resistance, sensitivity to endogenous hormones, hypothalamicpituitary-adrenal axis function abnormalities, nutritional deficiencies, glucose metabolism changes, or fluid and electrolyte imbalance [4,15,[20][21][22].…”
Section: Introductionmentioning
confidence: 99%
“…Women with LLBC are reported to have lower levels of this neuroactive steroid (Rapkin et al, 1997); but this is not always the case (Schmidt, Purdy, Moore, Paul, & Rubinow, 1994), and differences in responses to hormonal changes may be more important. There is also some evidence for a genetic component to LLBC (Condon, 1993;Kaur et al, 2004).…”
mentioning
confidence: 99%
“…It is estimated that a majority of women manifest some form of LLBC; about 75% have at least one symptom from the PMDD criteria for diagnosis list, the minimum for PMS classification (Kaur et al, 2004;Rapkin, Mikacic, & Moatakef-Imani, 2003). About 3-10% have the minimum of five symptoms from the list, the requirement for PMDD classification (Cenac, Maikibi, & Develoux, 1987;Hallman, 1986;Halbreich et al, 2003;Deuster, Adera, & South-Paul, 1999;Kaur et al, 2004).…”
mentioning
confidence: 99%
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