Predisposition to violence

Mednick, Sarnoff A.; Brennan, Patricia A.; Kandel, Elizabeth

doi:10.1002/1098-2337(1988)14:1<25::aid-ab2480140105>3.0.co;2-9

Cited by 34 publications

(20 citation statements)

References 15 publications

(2 reference statements)

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“…Violent offenders tended to have serious neuropsychological deficits, and those classified as brain damaged had significantly higher rates of violent behaviors than a nonbrain-damaged group. Weller (1986) presented evidence that left temporal or frontal lobe dysfunction is associated with violent behavior in persons diagnosed as suffering from psychopathy; and Mednick, Brennen, and Kandel (1988) cited evidence that violent offenders (juvenile and adult) showed greater frequency of brain damage than non-violent offenders. Further support for neurological factors in violence came from a follow-up study by Lewis, Lovely, Yeager, and Femina (1989) of 95 formerly incarcerated delinquents.…”

Section: This Research Was Funded In Part By a Grant From The Universmentioning

confidence: 99%

Quantitative EEG Findings Among Men Convicted of Murder

Evans¹,

Park²

1997

Journal of Neurotherapy

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Section: This Research Was Funded In Part By a Grant From The Universmentioning

confidence: 99%

Quantitative EEG Findings Among Men Convicted of Murder

Evans¹,

Park²

1997

Journal of Neurotherapy

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“…Consistent results for illicit drugs are more limited, but the findings generally support genetic contributions to the abuse of illicit drugs as well [Anthenelli and Schuckit, 1992;Cadoret et al, 1986;Grove et al, 1990]. Genetic factors have also been implicated for the development of depression [Kendler et al, 1993;Kendler et al, 1994;Wierzbicki, 1986] and antisocial behavior [Caspi and Moffitt, 1995;Cloninger and Gottesman, 1987;Dishion et al, 1995;Mednick et al, 1988;Moffitt, 1993;Rutter et al, 1990] in both clinical and epidemiological samples.…”

Section: Introductionmentioning

confidence: 99%

The familial aggregation of depressive symptoms, antisocial behavior, and alcohol abuse

et al. 1997

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This study describes results from an ongoing family study of adolescent boys and their families designed to investigate potential risk factors for substance abuse. The adolescent treatment probands have severe drug and alcohol related problems and were recruited through a residential rehabilitation program. To date, the sample includes 251 individuals: 39 male probands and their families and 34 control families matched for age and geographic location (zip code). Probands and participating family members are given a structured interview which assesses alcohol and drug problems, and various psychiatric symptoms. The purpose of the present study was to examine the coaggregation of depressive symptoms, antisocial behavior, and alcohol misuse. Multivariate pedigree analyses were performed using a model that allowed for the estimation of vertical familial transmission, residual sibling resemblance, and assortative mating. Spouse correlations were estimated at .57, .21, and .31 for antisocial behavior, depressive symptoms, and alcohol abuse, respectively. Residual sibling environment (i.e., sibling resemblance unaccounted for by parent-offspring transmission) was not found for alcohol problem symptoms, but did contribute to resemblance for antisocial behavior and depressive symptoms. The proportion of variance accounted for by vertical familial transmission was estimated at approximately 30 to 40%. More important, correlations among the transmissible family factors for these psychiatric syndromes ranged from .58 to .73, suggesting substantial overlap among the underlying familial antecedents for these disorders.

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“…Alguns estudaram a influência dos fato-res genéticos e contribuições fisiológicas (Brunner et al,1993;Christiansen, Knussmann, 1987;Dilalla, Gottesman, 1991;Loeber, Stouthamer-Loeber, 1998;Meddnick, Brenannan, Kandel, 1988;Renfrew, 1997). Outros examinaram o baixo desempenho escolar, baixa auto-estima e baixa expectativa (Gomide, 2000;Joffe, et al, 1990;Straus, 1994), ou o uso de substâncias tóxicas (Gomide, 2000;Renfrew, 1997;Stiffman et al, 1996;Tavares, Béria, Lima, 2001).…”

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