1996
DOI: 10.1016/0735-1097(95)00454-8 View full text |Buy / Rent full text
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Abstract: Predischarge 24-h mean heart rate is a strong predictor of mortality after myocardial infarction that can compete with left ventricular ejection fraction and heart rate variability.

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“…This has been shown, for example, in patients with congestive heart failure, in which indirect and direct markers of systemic and regional sympathetic drive (venous plasma norepinephrine, heart rate variability, heart rate spectral power, cardiac and renal norepinephrine spillover) have been documented to bear a close and direct relationship with cardiovascular mortality, disease progression and arrhythmic events, including sudden death (46)(47)(48)(49)(50)(51). Similar conclusions have been drawn by assessing the prognostic values of plasma norepinephrine or heart rate variability in the post-stroke phase following an acute myocardial infarction or in patients with vasospastic angina (52)(53)(54)(55). Finally, dysfunction in sympathetic cardiovascular control has also been shown to have prognostic value in non-cardiovascular diseases such as diabetes mellitus and renal failure (56,57), thereby documenting the clinical relevance of the neuroadrenergic overdrive in a broad spectrum of pathological states.…”
Section: Sympathetic Activation Cardiovascular Risk and Organ Damagementioning
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“…This has been shown, for example, in patients with congestive heart failure, in which indirect and direct markers of systemic and regional sympathetic drive (venous plasma norepinephrine, heart rate variability, heart rate spectral power, cardiac and renal norepinephrine spillover) have been documented to bear a close and direct relationship with cardiovascular mortality, disease progression and arrhythmic events, including sudden death (46)(47)(48)(49)(50)(51). Similar conclusions have been drawn by assessing the prognostic values of plasma norepinephrine or heart rate variability in the post-stroke phase following an acute myocardial infarction or in patients with vasospastic angina (52)(53)(54)(55). Finally, dysfunction in sympathetic cardiovascular control has also been shown to have prognostic value in non-cardiovascular diseases such as diabetes mellitus and renal failure (56,57), thereby documenting the clinical relevance of the neuroadrenergic overdrive in a broad spectrum of pathological states.…”
Section: Sympathetic Activation Cardiovascular Risk and Organ Damagementioning
“…73 In patients with cardiovascular disease, reduced heart rate variability is associated with poorer outcomes. [75][76][77] The Framingham Heart Study has implicated lower heart rate variability as a precursor to the development of future hypertension, 78 and increased BP variability has been implicated in increased risk of end-organ damage in patients with hypertension. 79 …”
Section: Cardiovascular Variabilitymentioning
“…Convincing data on association between tachycardia and unfavorable course of CVD have been received by now. HR is proved to be an independent predictor of development and complicated course of IHD, CHF and other CVDs [8][9][10][11][12][13][14][15]25,26]. In these studies all-causes mortality risk increased significantly in subjects with resting HR over 84 beats per min.…”
Section: Discussionmentioning