Posttraumatic cerebral infarction diagnosed by CT: prevalence, origin, and outcome.

Mirvis, Stuart E.; Wolf, Aizik L.; Numaguchi, Yuji; Corradino, G; Joslyn, John N.

doi:10.2214/ajr.154.6.2110744

Cited by 60 publications

(73 citation statements)

References 11 publications

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“…In our study, PTCI was a result of a focal mass effect and/or gross mechanical displacement of the brain, producing transfalcine or transtentorial herniation in all patients. In this study, infarction at the territory of PCA was the most common, consistent with the results of Mirvis et al 17,26) . Subfalcian herniation of the cingulate gyrus results in compression of one or both ACA or the callosomarginal artery and its branches 12,21) .…”

Section: Discussionsupporting

confidence: 93%

“…Sato et al 22) reported that the incidence of occipital lobe infarction was 9% among patients with transtentorial herniation on brain CT. Gross mechanical shift of the brain and herniation across the falx and/or tentorium accounted for most cases of infarction, ranging from 81% to 88% 17,24) . In our study, PTCI was a result of a focal mass effect and/or gross mechanical displacement of the brain, producing transfalcine or transtentorial herniation in all patients.…”

Section: Discussionmentioning

confidence: 99%

“…MCA territory infarction occurs due to a gross mass effect and herniation or severe brain swelling/edema 17,24,26) . Potential contributing factors include stretching and attenuation of the MCA, increased ICP, and a direct pressure effect from an extraaxial hematoma 17,24,28) .…”

Section: Discussionmentioning

confidence: 99%

“…nisms have been suspected for this complication, including cerebral vasospasm, vascular injury, embolization, and systemic hypoperfusion 8,10,13,17,19,21,22,24,26,27) . Malignant middle cerebral artery (MCA) infarction in a cerebral infarction in MCA territory was first described by Hacke 4) .…”

mentioning

confidence: 99%

See 3 more Smart Citations

Post-Traumatic Cerebral Infarction : Outcome after Decompressive Hemicraniectomy for the Treatment of Traumatic Brain Injury

Ham

Lee

Jang

et al. 2011

J Korean Neurosurg Soc

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Section: Discussionsupporting

confidence: 93%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

mentioning

confidence: 99%

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Post-Traumatic Cerebral Infarction : Outcome after Decompressive Hemicraniectomy for the Treatment of Traumatic Brain Injury

Ham

Lee

Jang

et al. 2011

J Korean Neurosurg Soc

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“…Thus, patients with concussion injury must be observed even though their first cerebral images are normal. Post-traumatic cerebral infarction is a known complication of craniocerebral trauma, with a frequency ranging from 1.9% to 10.4% [3,4]. It was suggested that cerebral vasospasm, direct vascular compression by mass effects, vascular injury, embolization, and systemic hypoperfusion play a role in the etiology [4].…”

mentioning

confidence: 99%

Post-Traumatic Late Onset Cerebral Ischemia

Genç¹

2015

Ann Clin Anal Med

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ÖzetTravma sonrası iskeminin en sık nedeni çoğunlukla diseksiyon nedeniyle ortaya çı-kan arterden artere emboli veya kraniyoservikal arterlerin tıkanmasıdır. 29 yaşın-daki erkek hasta kafa travmasının olmadığı sert paraşütle inişinden sonra 45 dakika süren bilinç kaybı nedeni ile 3 gün önce başka bir hastaneye başvurmuş ve nörolojik muayenesinin ve beyin BT incelemesinin normal olması üzerine 24 saatlik gözlem sonrası taburcu edilmiş. Taburcu olduktan 2 gün sonra epileptik nöbet geçiren hasta hastanemize başvurdu. Nörolojik muayenede sol hemianopi saptanan hastanın çekilen beyin MR incelemesinde sağ oksipital bölgede subakut iskemi saptandı. Romatolojik, kardiyolojik ve vasküler patolojiler dışlandıktan sonra hastaya geç başlangıçlı serebral iskemi tanısı konarak antiödem ve antiepileptik tedavi başlandı. Hasta sol hemianopi ve hafi kognitif bozukluk ile taburcu edildi. Travmayı takiben bilinç kaybı olan hastaların en az 72 saat gözlem altında tutulmasının mantıklı olacağını düşünmekteyiz. Anahtar KelimelerPost-Travmatik Serebral İskemi; Post-Travmatik Epilepsi; Geç Başlangıçlı Epilepsi; Kafa Travması Abstract Artery-to-artery emboli or occlusion of craniocervical arteries mostly due to dissection are the most common causes of ischemia after trauma. A 29 year-old male had been admitted to another hospital with loss of consciousness lasting for about 45 minutes after a hard parachute landing without head trauma three days ago. As his neurological examination and brain CT were normal, he had been discharged after 24 hours of observation. Two days after his discharge, he was admitted to our department with epileptic seizure. His neurological examination revealed left hemianopia. After observing occipital subacute ischemia at right side in brain magnetic resonance imaging (MRI), we performed cerebral angiography and no dissection was observed. Excluding the rheumatologic, cardiologic and vascular events, our final diagnosis was late onset cerebral ischemia. Anti-edema and antiepileptic treatment was initiated. He was discharged with left hemianopia and mild cognitive deficit. We suggest that it will be wise to hospitalize patients for at least 72 hours who has a history of unconsciousness following trauma.

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The role of serum Dickkopf‐1 in predicting 30‐day death in severe traumatic brain injury

et al. 2020

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Posttraumatic cerebral infarction diagnosed by CT: prevalence, origin, and outcome.

Cited by 60 publications

References 11 publications

Post-Traumatic Cerebral Infarction : Outcome after Decompressive Hemicraniectomy for the Treatment of Traumatic Brain Injury

Post-Traumatic Cerebral Infarction : Outcome after Decompressive Hemicraniectomy for the Treatment of Traumatic Brain Injury

Post-Traumatic Late Onset Cerebral Ischemia

The role of serum Dickkopf‐1 in predicting 30‐day death in severe traumatic brain injury

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