Post-Traumatic Pain and the Causalgic Syndrome

Doupe, J.; Cullen, Colden; Chance, G. Q.

doi:10.1136/jnnp.7.1-2.33

Cited by 168 publications

(29 citation statements)

References 13 publications

(10 reference statements)

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“…The condition may spread centrally from the stump to involve the phantom limb. Hyperalgesia and allodynia may be referred to the phantom limb upon stimulation of the stump, whether or not the stump is painful or shows signs of trophic or vascular changes (60). The characteristic qualities of superficial burning pain and deep aching pain may be additional evidence of the involvement of the sympathetic nervous system (60).…”

Section: Phantom Limb Painmentioning

confidence: 99%

Psychophysiological Contributions to Phantom Limbs

Katz

1992

Can J Psychiatry

117

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Section: Phantom Limb Painmentioning

confidence: 99%

Psychophysiological Contributions to Phantom Limbs

Katz

1992

Can J Psychiatry

117

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“…In all cases, the limb affected was the same lower extremity that was disabled by sciatic pain. 5. A male patient with foot drop, who did not undergo surgery, also developed thrombophlebitis.…”

Section: Clinical Materialsmentioning

confidence: 95%

Thrombophlebitis as a Complication of the Conservative Management of Herniated Lumbar Intervertebral Discs

1961

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“…The resulting peripheral vasoconstriction was supposed to induce tissue hypoxia, as the major nociceptive stimulus. In contrast, Doupe et al [ 4 ] postulated a pathologic generation of synapses between nociceptive afferents and sympathetic efferents. Impressed by therapeutic effi cacy of sympathetic blocks, Evans [ 5 ] created another name in 1946 ("sympathetic refl ex dystrophy"), which was also eventually dropped.…”

Section: Introductionmentioning

confidence: 94%

Evolving understandings about complex regional pain syndrome and its treatment

Fechir

Geber²,

Birklein³

2008

Current Science Inc

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Complex regional pain syndrome (CRPS) is still a puzzling disease. Although pathophysiologic understanding has improved, not every aspect of this challenging neuropathic pain syndrome has been explored. Typical symptoms of CRPS are sensory, motor, and autonomic dysfunctions. In most cases, CRPS occurs after a fracture, limb trauma, or lesion of the peripheral or central nervous system. Sometimes, symptoms develop without any trauma. Recent pathophysiologic concepts basically consider three major mechanisms: enhanced peripheral neurogenic inflammation, dysfunction of the sympathetic nervous system, and structural reorganization in the central nervous system. Moreover, a genetic predisposition may explain increased vulnerability. Treatment usually requires a multidisciplinary approach, including medical and nonmedical therapies. The common therapeutic aim is to maintain or restore normal function of the affected extremity. Beyond highlighting pathophysiologic concepts, this article describes recent therapeutic approaches.

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Post-Traumatic Pain and the Causalgic Syndrome

Cited by 168 publications

References 13 publications

Psychophysiological Contributions to Phantom Limbs

Psychophysiological Contributions to Phantom Limbs

Thrombophlebitis as a Complication of the Conservative Management of Herniated Lumbar Intervertebral Discs

Evolving understandings about complex regional pain syndrome and its treatment

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