Positive airway pressure therapy for heart failure

Kato, Takao; Suda, Shoko; Kasai, Takatoshi

doi:10.4330/wjc.v6.i11.1175

Cited by 44 publications

(28 citation statements)

References 139 publications

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“…CPAP improves airflow by maintaining upper airway patency, it also helps chest inflation. PAP increases the pharyngeal cross-sectional area, whereas in the intrathoracic compartment it facilitates recruitment of collapsed alveoli (3). Previous studies on genioglossus electromyographic activity during CPAP use in asleep adults suggest that the acute effect of extrathoracic airway stenting is passive (4,5).…”

Section: Ventilatory Effectsmentioning

confidence: 97%

“…An increase in the positive end-expiratory pressure (PEEP) by increasing lung volume decreases venous return by a diminished pressure gradient. This leads to decelerating venous blood flow, decreased RV filling and, consequently, diminished RV stroke volume (3).…”

Section: Right and Left Ventricular Functionmentioning

confidence: 99%

“…The effect of increased airway pressure on the cardiorespiratory system Since its invention, CPAP has been used as effective treatment in cardiogenic pulmonary oedema, obstructive sleep apnoea (OSA) and in adult respiratory distress syndrome (ARDS). Positive airway pressure (PAP) causes an increased chest inflation, diminishes the development of atelectasis, recruits collapsed alveoli, decreases airway resistance, it reduces inspiratory effort and decreases of work of breathing (3).…”

Section: Introductionmentioning

confidence: 99%

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Cardiorespiratory interaction with continuous positive airway pressure

Pengo¹,

Bonafini²,

Fava³

et al. 2018

J. Thorac. Dis.

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The treatment of choice for obstructive sleep apnoea (OSA) is continuous positive airway pressure therapy (CPAP). Since its introduction in clinical practice, CPAP has been used in various clinical conditions with variable and heterogeneous outcomes. In addition to the well-known effects on the upper airway CPAP impacts on intrathoracic pressures, haemodynamics and blood pressure (BP) control. However, short- and long-term effects of CPAP therapy depend on multiple variables which include symptoms, underlying condition, pressure used, treatment acceptance, compliance and usage. CPAP can alter long-term cardiovascular risk in patients with cardiorespiratory conditions. Furthermore, the effect of CPAP on the awake patient differs from the effect on the patients while asleep, and this might contribute to discomfort and removal of the use interface. The purpose of this review is to highlight the physiological impact of CPAP on the cardiorespiratory system, including short-term benefits and long-term outcomes.

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Section: Ventilatory Effectsmentioning

confidence: 97%

Section: Right and Left Ventricular Functionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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Cardiorespiratory interaction with continuous positive airway pressure

Pengo¹,

Bonafini²,

Fava³

et al. 2018

J. Thorac. Dis.

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“…In ADHF, PAP therapy increases oxygenation through the recruitment of collapsed alveoli, induces fluid shifts back from alveoli and interstitial space to the pulmonary circulation, reduces respiratory muscle load and the work of breathing, and stabilizes hemodynamics [105]. …”

Section: Acid–base Abnormalities In Chfmentioning

confidence: 99%

Acid–base and electrolyte abnormalities in heart failure: pathophysiology and implications

2015

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Electrolyte and acid–base abnormalities are a frequent and potentially dangerous complication in subjects with congestive heart failure. This may be due either to the pathophysiological alterations present in the heart failure state leading to neurohumoral activation (stimulation of the renin–angiotensin–aldosterone system, sympathoadrenergic stimulation), or to the adverse events of therapy with diuretics, cardiac glycosides, and ACE inhibitors. Subjects with heart failure may show hyponatremia, magnesium, and potassium deficiencies; the latter two play a pivotal role in the development of cardiac arrhythmias. The early identification of these alterations and the knowledge of the pathophysiological mechanisms are very useful for the management of these patients.

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“…Our objectives are as follows: (1) elucidating the therapeutic effects of catheter ablation and implantable cardioverter defibrillators in patients with atrial or ventricular arrhythmia, (2) investigating the efficacy of cardiac resynchronization therapy in patients with advanced heart failure, and (3) …”

Section: ) Gaku Sekita (Associate Professor)mentioning

confidence: 99%

Department of Cardiovascular Medicine

2015

Juntendo Medical Journal

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We have conducted a wide spectrum of basic and clinical research to elucidate the pathophysiology of and develop novel techniques of diagnosis and therapeutic strategies for cardiovascular diseases. Basic research involves cardiovascular physiology, pathology, molecular and vascular biology, and lipidology. Clinical research involves epidemiology and studies that occur in a clinical laboratory, and clinical trials. We believe that our research findings are contributing to evidence-based health care in the field of cardiovascular medicine.

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Positive airway pressure therapy for heart failure

Cited by 44 publications

References 139 publications

Cardiorespiratory interaction with continuous positive airway pressure

Cardiorespiratory interaction with continuous positive airway pressure

Acid–base and electrolyte abnormalities in heart failure: pathophysiology and implications

Department of Cardiovascular Medicine

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