Poor saccadic control correlates with dyslexia

Egocentric mental rotation in Hungarian dyslexic children

“…According to this result, one of the outputs from the magnocellular system goes to the parietal cortex besides the primary visual system. Biscaldi et al (1998) demonstrated abnormal saccade control in dyslexic children. They speculated that the parietal cortex might be the common neural substrate for the differences in saccade performance between dyslexic and control children.…”

Wearable eye tracking for mental health monitoring

“…Abnormal eye movements have been found in dyslexic children and for some time researchers wondered whether it were impaired eye movements that were causing dyslexia. Nowadays the theory tends to be that it is dyslexia itself that is causing abnormal eye movement patterns in dyslexic children, although this is not widely accepted amongst researchers [27]. It has been shown that children with dyslexia show longer fixations and increased regressions in reading [28] and Biscaldi, Gezeck and Stuhr found a correlation between abnormal saccadic control and reading disabilities when studying 185 persons [27].…”

“…Nowadays the theory tends to be that it is dyslexia itself that is causing abnormal eye movement patterns in dyslexic children, although this is not widely accepted amongst researchers [27]. It has been shown that children with dyslexia show longer fixations and increased regressions in reading [28] and Biscaldi, Gezeck and Stuhr found a correlation between abnormal saccadic control and reading disabilities when studying 185 persons [27]. Monitoring eye movements on a daily basis or over long periods of time could potentially give researchers new insights on dyslexia.…”

Why do words jump? An exploration of visually symptomatic readers

“…It is thus likely that removal of these visual barriers encourages learners to read for longer and it may be that reading becomes more pleasurable, leading to more practice, which in turn culminates in better reading. The impact of any visual anomaly may also be greater in poor or less able readers since they read in a different way, with more fixations, greater time spent reading and longer duration of fixations, with longer latency [67][68][69] (visual behaviours that may make the spatial aspects of text more obvious). Able readers make fewer fixations, take less time to read and skim read, [67][68][69] making the demands on the visual system less arduous and possibly the pattern effects less obvious or distracting.…”

“…The impact of any visual anomaly may also be greater in poor or less able readers since they read in a different way, with more fixations, greater time spent reading and longer duration of fixations, with longer latency [67][68][69] (visual behaviours that may make the spatial aspects of text more obvious). Able readers make fewer fixations, take less time to read and skim read, [67][68][69] making the demands on the visual system less arduous and possibly the pattern effects less obvious or distracting. 47 In conclusion, assessing the visually symptomatic reader requires thorough history-taking, modified clinical examination and a methodological approach to assessment and differential diagnosis.…”