Polymyalgia rheumatica, temporal arteritis and malignancy

Speed, C. A.; Haslock, I

doi:10.1136/pgmj.71.838.500

Cited by 10 publications

(3 citation statements)

References 9 publications

(1 reference statement)

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“…Rarely, both polymyalgia rheumatica and Horton's arteritis are associated with lung neoplasm or hematological disorders (i.e., macroglobulinemia of Waldenstrom) [ 30 ].…”

Section: Clinical Characteristics and Diagnosismentioning

confidence: 99%

The Thromboembolic Risk in Giant Cell Arteritis: A Critical Review of the Literature

Guida

Tufano

Perna

et al. 2014

International Journal of Rheumatology

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Giant cell arteritis is a systemic vasculitis characterized by granulomatous inflammation of the aorta and its main vessels. Cardiovascular risk, both for arterial and venous thromboembolism, is increased in these patients, but the role of thromboprophylaxis is still debated. It should be suspected in elderly patients suffering from sudden onset severe headaches, jaw claudication, and visual disease. Early diagnosis is necessary because prognosis depends on the timeliness of treatment: this kind of arteritis can be complicated by vision loss and cerebrovascular strokes. Corticosteroids remain the cornerstone of the pharmacological treatment of GCA. Aspirin seems to be effective in cardiovascular prevention, while the use of anticoagulant therapy is controversial. Association with other rheumatological disease, particularly with polymyalgia rheumatica is well known, while possible association with antiphospholipid syndrome is not established. Large future trials may provide information about the optimal therapy. Other approaches with new drugs, such as TNF-alpha blockades, Il-6 and IL-1 blockade agents, need to be tested in larger trials.

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“…Rarely, both polymyalgia rheumatica and Horton's arteritis are associated with lung neoplasm or hematological disorders (i.e., macroglobulinemia of Waldenstrom) [ 30 ].…”

Section: Clinical Characteristics and Diagnosismentioning

confidence: 99%

The Thromboembolic Risk in Giant Cell Arteritis: A Critical Review of the Literature

Guida

Tufano

Perna

et al. 2014

International Journal of Rheumatology

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Section: Tratamiento Y Pronósticounclassified

“…Las neoplasias pueden presentarse con síntomas similares a la ACG (81,82). La persistencia de los síntomas de ACG y/o la no normalización de los reactantes de fase aguda con la corticoterapia debe hacer considerar otros diagnósticos, entre los que se incluyen fundamentalmente las neoplasias (81). En una serie de pacientes con ACG hubo un aumento de la incidencia de cáncer, aunque el largo intervalo de tiempo entre el diagnóstico de ambas entidades hace improbable que la ACG pueda ser en algunos casos un síndrome paraneoplásico (82).…”

Section: Tratamiento Y Pronósticounclassified

Arteritis de células gigantes

Romero

2002

An. Med. Interna (Madrid)

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RESUMEN La arteritis de células gigantes (ACG) o arteritis temporal es la vasculitis sistémica más frecuente en nuestro medio, y su incidencia puede haber aumentado en los últimos años. La ACG afecta a vasos de mediano y grueso calibre, con preferencia por las arterias extracraneales, y ocurre en mayores de 50 años. La etiopatogenia es desconocida, aunque diversos agentes infecciosos pueden estar implicados. Una biopsia de arteria temporal normal no excluye el diagnóstico de ACG, ya que las lesiones pueden ser segmentarias. Las complicaciones más temibles de la ACG son la pérdida visual permanente, los ictus isquémicos y los aneurismas de aorta torácica y abdominal. El tratamiento consiste en corticoides a dosis altas, y actualmente no existe una alternativa terapéutica eficaz sin efectos adversos importantes. La mortalidad de los pacientes con ACG tratados no parece estar aumentada significativamente, probablemente debido al correcto diagnóstico y manejo de esta entidad. PALABRAS CLAVE: Arteritis de células gigantes. Arteritis temporal.

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Autoimmune paraneoplastic syndromes associated to lung cancer: A systematic review of the literature

et al. 2017

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Polymyalgia rheumatica, temporal arteritis and malignancy

Abstract: 1 Nestel PJ, Billington T. Effects of probucol on low density lipoprotein removal and high density lipoprotein synthesis. Atherosclerosis 1981; 38: 203-

Cited by 10 publications

References 9 publications

The Thromboembolic Risk in Giant Cell Arteritis: A Critical Review of the Literature

The Thromboembolic Risk in Giant Cell Arteritis: A Critical Review of the Literature

Arteritis de células gigantes

Autoimmune paraneoplastic syndromes associated to lung cancer: A systematic review of the literature

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