“…We confirmed that the secretion of amylase to external media is under autonomic nervous control, since all adrenergic and cholinergic agonists increased amylase activity in this media, being the β adrenergic agonist, isoproterenol, the most potent stimulant. Precisely, Quissell [16] reported that noradrenaline activates β adrenergic receptors leading to increased cAMP levels that is essential for exocytotic secretion. It has also been documented that stimulation of α adrenergic receptors or muscarinic receptors also elicits amylase release but at levels that are significantly lower than those observed from β adrenergic receptor-mediated response in rat parotid glands [14].…”