Physiologic specialization in Puccinia coronata avenae (Corda) Eriks and Henn

Abstract

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Characterization in isogenic lines of oat crown rust resistance genes from four sources

“…In general, younger leaves on the same plant are as susceptible as seedlings, but the older leaves are resistant (Hassebrauk, 1932;Newton and Brown, 1934;Murphy, 1935)* These observations are contrary to what had been reported for leaf rust of wheat (Johnston and Melchers,I929). Simons (1961) reported that some cultlvars of oats were resistant as adult plants to certain races of crown rust but susceptible to others.…”

“…Shortening of the incubation period at higher temperatures has been well documented (Marland, 1937;Peturson, 1930)• Shifts in infection type at higher temperatures are not un common (Murphy, 1935;Saari and Moore, 1962;Simons, 195^;Putrell and Rivers, 1955). The fundamental reactions in the breakdown of resistance are unknown; however, at least three schools of thought prevail: carbohydrate metabolism, in hibitory mechanisms, and molecular aspects of temperature response.…”

“…Murphy (1935) concluded that there was little effect of age of the oat plant on crown rust reactions from the seedling to the juvenile stages. After the juvenile stage, no considerable effect of age was noticed in his material.…”

Genetic analysis of seedling resistance to crown rust in five diploid oat (Avena strigosa) accessions

“…The seedlings were then placed in a misting chamber at room temperature for 24 hours, and later transferred to a growth room maintained at 23˚C. Two weeks later, seedlings were scored for crown rust response using an IT scale of "0"-"4" described by Murphy (1935), with minor modifications (refer Cabral and Park, 2014). While IT scores between "0" and "2" were considered resistant, those between "2-3" and "3" were indicative of moderate resistance, and scores of "3-3+" and "3+" indicated susceptibility.…”

Influence of Formulation on Efficacy of Three Systemic Fungicides for Control of Oat Leaf Rust

“…Hagborg (1971) was determined by summing the products of, the number of disease lesions and the infection type, 0 to 3 as described by Murphy (1935 For personal use only. As mentioned previously (Hagborg 1970 (Spiertz 1973 …”