Peptidergic modulation of the vagal effect on the cardiac rhythm

Pokrovskii, V. M.; Osadchii, Oleg E.; Курзанов, А. Н.

doi:10.1007/bf00840494

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Bulletin Of Experimental Biology and Medicine No-i2 1995 G1

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1995

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“…These factors probably contribute to the diminished vagal bradycardia after a course of TES. A role in limiting the bradycardia may also be played by the opioidergic system, which is activated after TES and whose modulating influences have been shown to limit not only adrenergic [2] but also cholinergic [9,10] activation. However, more research is needed to clarify the mechanisms of cardiac protection afforded by TES in myocardial infarction.…”

Section: Bulletin Of Experimental Biology and Medicine No-i2 1995 Gmentioning

confidence: 99%

A preliminary course of transauricular electrostimulation mitigates impairment of cardiac contractile function in rats with myocardial infarction

et al. 1995

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A course of transauricular electrostimulation (TES) consisting of 10 sessions was administered to rats before the induction of myocardial infarction by Selye's method and to rats that were left intact. In the latter animals, the electrostimulation did not influence cardiac contractile function at rest (as judged by heart rate, developed pressure, and Katz's index), but exerted beneficial chronotropic and inotropic effects during the maximum isometric tension produced by compression of the ascending aorta. In the TES-pretreated rats with a 2-day-old myocardial infarct, cardiac contractile function was depressed significantly less, both at rest and during isometric tension, than in infarcted rats not exposed to TES.

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Section: Bulletin Of Experimental Biology and Medicine No-i2 1995 Gmentioning

confidence: 99%