Parkinsonism as a Complication of Bariatric Surgery

Kamel, Walaa A.; Hashel, Jasem Y. Al; Kilany, Ayman; Altailji, Samira

doi:10.3889/oamjms.2015.121

Cited by 1 publication

(1 citation statement)

References 14 publications

(12 reference statements)

Supporting

Mentioning

Contrasting

Order By: Relevance

“…39 Four cases of parkinsonism developed acutely or lately following bariatric surgery have been identified. 40,41 An 18-years old female experiencing generalized seizure and ischemic stroke 4 months after surgery have been reported. 42…”

Section: Early Complicationsmentioning

confidence: 99%

When General Surgeon Should Call Neurologist and What Neurologist Should Know About Obesity Surgery?

Demirci¹,

Ünlü²

2020

Turkiye Klinikleri J Med Sci

View full text Add to dashboard Cite

ABS TRACT Obesity is an ever-increasing problem all over the world and may associate with and cause many metabolic conditions. Bariatric surgery is recognized as the most effective procedure in losing weight, maintaining the weight loss over time and in reversing the associated comorbidities. However, surgery itself may cause some systemic complications due to limited food and micronutrient passage through the operated stomach. Up to 16% of the patients may develop complications involving any part of the nervous system. Neurological complications may develop immediately or long years after the surgery. Immediate post-operative complications are mononeuropathy/plexopathy and rhabdomyolisis. The common early complications are encephalopathy (in the form of Wernicke or Korsakoff encephalopathy), radiculoneuropathy mimicking Guillain-Barre syndrome and polyneuropathy. Optic neuropathy, myelopathy, peripheral polyneuropathy may develop over longtime course. Patients with these neurological complications are found to have isolated or multiple nutrient deficiencies of vitamins and minerals. Among all thiamine, B12 and copper deficiency ranks as the most common ones observed after surgery. Deficiencies of riboflavin, niacin, pyridoxine, folate, vitamin D, and vitamin E, iron, calcium, phosphorus, magnesium and trace elements such as zinc, iodine, and selenium have also been reported. Symptoms vary according to presentation time, anatomical location and injury mechanism. Occurrence of these neurological complications emphasizes the importance of patient education about adherence to nutritional suggestions and surveillance after surgery. Awareness and early intervention may prevent or heal these disabling and sometimes fatal complications.ÖZET Obezite tüm dünyada giderek artan bir sorundur ve birçok metabolik bozukluğa neden olur veya bunlarla birlikte bulunabilir. Bariatrik cerrahi kilo vermede, zaman içinde kilo kaybını korumada ve ilişkili komorbiditeleri tersine çevirmede en etkili yöntem olarak kabul edilir. Bununla birlikte, ameliyatın kendisi ameliyat edilen mideden sınırlı yiyecek ve mikro besin geçişi nedeniyle bazı sistemik komplikasyonlara neden olabilir. Hastaların %16 kadarında sinir sisteminin herhangi bir bölümünü etkileyen komplikasyonlar gelişebilir. Nörolojik komplikasyonlar cerrahiden hemen sonra ya da yıllar sonra ortaya çıkabilir. Cerrahi sonrası hemen gelişen komplikasyonlar mononöropati/ pleksopati ve rabdomiyolizdir. Sık erken komplikasyonlar ensefalopati (Wernicke veya Korsakoff ensefalopatisi şeklinde), Guillain-Barre sendromunu taklit eden radikulonöropati ve polinöropatidir. Optik nöropati, miyelopati ve periferik polinöropati uzun dönemde gelişebilir. Bu nörolojik komplikasyonları olan hastalarda tek veya çoklu vitamin ve mineral eksiklikleri saptanabilir. Tiamin, B12 ve bakır eksikliği cerrahi sonrası en sık görülenlerdir. Riboflavin, niasin, piridoksin, folat, vitamin D ve vitamin E, demir, kalsiyum, fosfor, magnezyum ve çinko, iyot ve selenyum gibi eser elementlerin eksikliği de bildirilmişt...

show abstract

Section: Early Complicationsmentioning

confidence: 99%