On the stress-induced photon emission from organism: II, how will the stress-transfer kinetics affect the photo-genesis?

Abstract

Much remains to be identified for the temporal course of stress-induced photon emission (PE) following stress of various types including but not limited to light. Induced PE often decays hyperbolically; yet, it is not uncommon for induced PE to manifest decay patterns that are various combinations of first-order responses. Induced PE also presented transient patterns characteristic of second-order responses. A soliton-based photon-storage model addressed the hyperbolic decay pattern of induced PE; however, there are questions regarding non-hyperbolic decay as well as the large range of delayand-decay scales of induced PE. This work offers an alternative interpretation of the temporal course of induced PE when stressed upon an organism. It is proposed that the surface photon emission of induced PE due to a stress involves two causally sequential phases: a stress-transfer phase that transforms the stress to photo-genesis, and a photon-propagation phase that transmits the photons from the site of photo-genesis to surface emission. Part I has argued that a retarded or slow stress-transfer phase is necessary to explain induced PE occurring/lasting at a timescale several orders of magnitude later/longer than the photon propagation delay due to tissue scattering after stress-removal. Part II models the kinetics of the stress-transfer phase that sources the photo-genesis with a linear-system approach. The analysis illustrates how a single stress-transfer pathway may manifest various photo-genesis patterns in responding to the same stressinput, and why a single kinetic pattern of photo-genesis may arise from multiple paths of stress transfer. The theoretical insights may help devise stress-control strategies to enhance the yield of induced PE for more mechanistic discoveries and potentiating broader applications.

Phenomenological Interpretations of Some Somatic Temporal and Spatial Patterns of Biophoton Emission in Humans

“…This will require treating the photon diffusion problem as a strictly temporal problem with fast time-responses, not the steady-state problem with slower temporal modulation of the photo-genic source as is simplified in this part of the work for superficial profiling of steadystate spontaneous biophoton emission. Works based on time-domain photon propagation approaches have been demonstrated by the author to address the kinetics of the changes of biophoton emission induced by external stress that exerts significantly faster modulation to the photogenesis (Piao, 2020a;Piao, 2020b). The theoretical outcomes of those works agree with what others have observed (Chwirot, 1988;Hagens et al, 2008) and seem to support the central hypothesis of this work.…”

Biophotons and Emergence of Quantum Coherence—A Diffusion Entropy Analysis

“…The advocates of quantum mechanics to explain the origin of cognition may use these results to support their view. We limit ourselves to notice that other conjectures may be invoked, for instance the influence of stress on the biophoton emission [ 63 , 64 ]. In this case, the stress may be related to the fact that the seedlings begin to grow in an environment completely devoid of light.…”

“…However, we do not rule out the connection with self-organized criticality that may be the source of the joint action of the infinite FBM memory and crucial events [ 53 ]. We shall explore this important issue by comparing the transition to FBM induced by autonomic neuropathy disease in human beings [ 53 ] to the hypotheses of stress signaled by biophoton emission [ 63 , 64 ]. We hope that the present paper may contribute to shedding further light on the role played by biophotons on these processes.…”

JSE 35:2 Summer 2021

“…( 11) and ( 12) will infer the age-dependency of the temporal differentiation of by setting = 1 and = 0, the effect of systemic physiology over a time period by setting = 0, and the outcome of acquired pathology at a given time point by setting = 1. We further hypothesize the vivodensity to be proportional to a chemical potential of the metabolic aspect (Piao, 2020b) which may regulate how a systemic perturbation to metabolism is transferred to a local photo-genesis process to cause observable temporal changes in biophoton emission, through the oxidative bio-chemical pathways that produce free radicals with ROS (Slawinski et al, 1992;Burgos et al, 2016). And this chain reaction can be treated as the changing rate of "vivodensity" that modulates oxidative stress.…”

“…The analysis speculates that there is a collective state of autonomic neuro-energy expressed in a form of "vivodensity," which in essence is the maximal coherence among the energy "modes" of neuronal control that an intelligent being represented by humans is able to engage in at any given time of life. This vivodensity could also be considered as being proportional to a chemical potential of the metabolic aspect (Piao, 2020b) which regulates how a systemic perturbation to metabolism is transferred to a local photo-genesis process to cause observable temporal changes in biophoton emission. This vivodensity thus is treated to modulate metabolic activities responsible for photo-genesis pertinent to biophotons.…”