2019
DOI: 10.1007/s11910-019-0937-8
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Occipital Neuralgia and Cervicogenic Headache: Diagnosis and Management

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Cited by 65 publications
(81 citation statements)
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“…6). 9 The cervical neck strain from donning the equipment could have led to the development of cervicogenic headache or tension-type headache (TTH) [22][23][24] The peripheral sensitisation may activate the trigeminocervical complex through nociceptive information transmitted via different branches of the trigeminal nerve through the trigeminal ganglia and brainstem to the higher cortical areas thereby triggering the headache attacks coronavirus disease 2019 25,26 Alternatively, a neuralgia with transient effects on the underlying superficial sensory nerves could have occurred, although other reasons were more likely as most respondents did not report characteristics suggestive of a neuropathic process. 27 These etiological reasons could perhaps explain why a large proportion of those with pre-existing primary headache disorders and concomitant de novo PPE-associated headaches reported an increase in the average number of headache days over a 30 day period, with the perception that this change was probably attributable to the PPE.…”
Section: Discussionmentioning
confidence: 99%
“…6). 9 The cervical neck strain from donning the equipment could have led to the development of cervicogenic headache or tension-type headache (TTH) [22][23][24] The peripheral sensitisation may activate the trigeminocervical complex through nociceptive information transmitted via different branches of the trigeminal nerve through the trigeminal ganglia and brainstem to the higher cortical areas thereby triggering the headache attacks coronavirus disease 2019 25,26 Alternatively, a neuralgia with transient effects on the underlying superficial sensory nerves could have occurred, although other reasons were more likely as most respondents did not report characteristics suggestive of a neuropathic process. 27 These etiological reasons could perhaps explain why a large proportion of those with pre-existing primary headache disorders and concomitant de novo PPE-associated headaches reported an increase in the average number of headache days over a 30 day period, with the perception that this change was probably attributable to the PPE.…”
Section: Discussionmentioning
confidence: 99%
“…Despite this relatively strict criterion, it can be difficult to clinically diagnose CGHA. For instance sharp pain in the occipital region may reflect occipital neuralgia, which can mimic a CGHA (Barmherzig & Kingston, 2019), whereas overlapping signs and symptoms with other forms of headaches can imitate CGHA, including neck pain, nausea, vomiting, photophobia and phonophobia associated with migraine headaches (Fredriksen, Antonaci, & Sjaastad, 2015;Sjaastad, 2008).…”
Section: Introductionmentioning
confidence: 99%
“…Moreover, Janis et al (2010), reported six major compression points along its course in 25 fresh cadaveric heads which were: between the SSC and OCI, at the entrance and the exit from the SSC, at the entrance and exit from the TP and at the crossing point with the occipital artery [11]. Treatment options of ON include posture correction, pharmacological treatment, local anesthetic injection, botulinum toxin infiltrations, pulse radiofrequency therapy and surgery [1,5]. Occipital nerve block using a local anesthetic with corticosteroids was injected at 2 cm lateral and 2 cm inferior to the external occipital protuberance (EOP) for blocking the GON [13].…”
Section: Introductionmentioning
confidence: 99%