Nutritional Requirements and Feeding Recommendations for Small for Gestational Age Infants

Tudehope, David; Vento, Máximo; Bhutta, Zulfiqar A.; Pachi, Paulo Roberto

doi:10.1016/j.jpeds.2012.11.057

Cited by 66 publications

(47 citation statements)

References 48 publications

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“…Consequently, the fetus has less lean mass, less body fat, lower bone mineral content, and lower glycogen stores, and is at increased risk for hypoglycemia at birth. [12][13][14] Although preterm SGA infants are considered to require a higher energy supply than appropriate for GA infants, enteral feeding guidelines are not well established for preterm SGA neonates, and no published information on the optimal feeding regimen is available. 5 Our randomized controlled trial assessed the efficacy of a PFR in reducing the hospital LOS in a population of moderately preterm SGA infants.…”

Section: Discussionmentioning

confidence: 99%

Proactive Enteral Nutrition in Moderately Preterm Small for Gestational Age Infants: A Randomized Clinical Trial

Zecca

Costa

Barone

et al. 2014

The Journal of Pediatrics

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Section: Discussionmentioning

confidence: 99%

Proactive Enteral Nutrition in Moderately Preterm Small for Gestational Age Infants: A Randomized Clinical Trial

Zecca

Costa

Barone

et al. 2014

The Journal of Pediatrics

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“…24 They often consider preterm and growth-restricted neonates in the single category of low BW infants, although these pathological conditions are different. 25 A standard, albeit controversial, 26,27 goal for these babies has been weight catch-up by 1 year of age to improve neurological development.…”

Section: Perspectivesmentioning

confidence: 99%

Rapid Early Growth May Modulate the Association Between Birth Weight and Blood Pressure at 5 Years in the EDEN Cohort Study

et al. 2016

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Abstract-Physiological evidence suggests that birth weight (BW) and postnatal growth affect blood pressure (BP) level, independently or in interaction. Their respective roles are difficult to disentangle in epidemiological studies, however, especially when adjusting for final weight. We assessed the portion of the effect of BW on BP at 5 years that was not attributable to postnatal growth and investigated potential interactions between BW and postnatal growth velocity at different time points in the EDEN mother-child study. Collecting a median of 19 weight measurements for each of the 1119 children who completed follow-up enabled us to model instantaneous growth velocity at any age. After computing a BP SD-score at 5 years, adjusted for age, sex, current body mass index, and height, we used multiple linear regression to study its association with age-and sex-specific BW z score, adjusting for several maternal and pregnancy risk factors. We tested interactions between BW categories (small-, appropriate-, and large-for-gestational-age) and weight growth velocities at different ages. The BW z score was negatively and significantly correlated with the systolic BP SD-score at the age of 5 years (r=−0.07, P=0.02). Interactions were found between BW categories and weight growth velocities from 1 to 4 months (P from 0.002 to 0.08) but not at older ages; specifically, children born small for gestational age with a fast weight growth velocity in their first few months of life had the highest absolute systolic BP and SD score values at 5 years. They may need monitoring for cardiovascular risks. (Hypertension. 2016;68:859-865.

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“…Similarly, a delay in mammary gland maturation during late pregnancy would be a possible consequence of reduced CX26 expression/function, whereas CX43 deficiency would be expected to blunt the effect of oxytocin in inducing postnatal milk extrusion. The seriousness of this latter effect could be compounded if, as in the mouse (Winterhager et al 2013), a reduced level of CX43 in the decidual tissue results in intrauterine growth restriction, a condition that increases the importance of breastfeeding (Bozzetti et al 2013, Tudehope et al 2013. Such scenarios are not merely hypothetical, given that numerous human diseases and disabilities have been linked with mutations in connexin genes (Dobrowolski & Willecke 2009), including those encoding connexins mentioned in this review.…”

Section: Discussionmentioning

confidence: 97%

Physiological roles of connexins in labour and lactation

Kidder

Winterhager

2015

Reproduction

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The connexin family of proteins are best known as oligomerizing to form intercellular membrane channels (gap junctions) that metabolically and ionically couple cells to allow for coordinated cellular function. Nowhere in the body is this role better illustrated than in the uterine smooth muscle during parturition, where gap junctions conduct the contraction wave throughout the tissue to deliver the baby. Parturition is followed by the onset of lactation with connexins contributing to both the dramatic reorganization of mammary gland tissue leading up to lactation and the smooth muscle contraction of the myoepithelial cells which extrudes the milk. This review summarizes what is known about the expression and roles of individual connexin family members in the uterus during labour and in the mammary glands during development and lactation. Connexin loss or malfunction in mammary glands and the uterus can have serious implications for the health of both the mother and the newborn baby.Reproduction (2015) 150 R129-R136

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Nutritional Requirements and Feeding Recommendations for Small for Gestational Age Infants

Cited by 66 publications

References 48 publications

Proactive Enteral Nutrition in Moderately Preterm Small for Gestational Age Infants: A Randomized Clinical Trial

Proactive Enteral Nutrition in Moderately Preterm Small for Gestational Age Infants: A Randomized Clinical Trial

Rapid Early Growth May Modulate the Association Between Birth Weight and Blood Pressure at 5 Years in the EDEN Cohort Study

Physiological roles of connexins in labour and lactation

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