Myocardial Infarction in Thyrotoxicosis

Burstein, Josef; Lamberg, B.‐A.; Erämaa, Eino

doi:10.1111/j.0954-6820.1960.tb17392.x

Cited by 27 publications

(3 citation statements)

References 40 publications

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“…Angina pectoris has been reported to occur in 0.5%-20% (usually 10%-12%) of patients with thyrotoxicosis (12). Burstein et al investigated 384 cases of thyrotoxicosis, found 7 cases of typical AMI (all cases were Graves' disease including one patient who developed thyrotoxicosis after radioactive iodine [RAI] treatment), and reported that the incidence of CAD in patients with thyrotoxicosis was 3.9% (n = 15) (12).…”

Section: Discussionmentioning

confidence: 99%

Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Kim

Jung

Hahm

et al. 2011

Thyroid

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Section: Discussionmentioning

confidence: 99%

Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Kim

Jung

Hahm

et al. 2011

Thyroid

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“…Classical angina of effort is documented, but rare, in thyrotoxic patients with coronary artery disease and may lead to myocardial infarction 2,3 . Thyrotoxic patients may also have anginal symptoms without demonstrable coronary artery disease.…”

Section: Discussionmentioning

confidence: 99%

“…Thyrotoxicosis may exacerbate angina and can even precipitate myocardial infarction in patients with pre‐existing coronary artery disease 1–3 . Presumably myocardial ischaemia is worsened by the increased cardiac workload which occurs in hyperthyroidism.…”

mentioning

confidence: 99%

Variant Angina in Thyrotoxicosis

Phull

Collins

Norell

et al. 1993

Int J Clinical Practice

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SUMMARYThyrotoxicosis may exacerbate angina pectoris in patients with coronary artery disease but angina also occurs in thyrotoxic patients with normal coronary arteries. Four female severely thyrotoxic patients presented with apparent angina pectoris as a manifestation of their thyrotoxicosis. Two of them had transiently abnormal ECGs during pain. Treatment for thyrotoxicosis immediately stopped the chest pain, which did not recur. All patients had negative exercise ECGs, and one had a normal coronary angiogram.The possible mechanisms to explain this phenomenon are discussed. Coronary artery spasm has been demonstrated in thyrotoxic patients previously and may explain the features in these patients. An alternative hypothesis is that myocardial metabolism is fundamentally changed by thyrotoxicosis. We would recommend that thyroid function assessment should be considered in young female patients with atypical angina.

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Angina in thyrotoxicosis. Thyroid-related coronary artery spasm

Featherstone¹

1983

Archives of Internal Medicine

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Ischemic heart disease is a common complication of thyrotoxicosis, although the exact mechanism has not been defined. A case is reported of angina and thyrotoxicosis in which the angina was reproduced by coronary artery spasm induced by ergonovine maleate, relieved by thyroid ablation, and reactivated by postablative thyroid replacement. Coronary arteriography did not show significant stenoses prior to ergonovine injection. Possible mechanisms of coronary artery spasm in thyrotoxicosis are briefly discussed. Hyperthyroidism should be considered as a cause of angina in any patient with rest pain and normal coronary arteries.

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Myocardial Infarction in Thyrotoxicosis

Cited by 27 publications

References 40 publications

Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Variant Angina in Thyrotoxicosis

Angina in thyrotoxicosis. Thyroid-related coronary artery spasm

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