Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Kim, Hee Jin; Jung, Tae Sik; Hahm, Jong Ryeal; Hwang, Seok-Jae; Lee, Sang Min; Jung, Jung Hwa; Kim, Sang-Yong; Chung, Soon Ii

doi:10.1089/thy.2010.0428

Cited by 18 publications

(18 citation statements)

References 16 publications

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“…10 Patients who experience AMI induced by thyrotoxicosis have an excellent prognosis. 4 The patient described in this report remained free of angina chest pain after taking nitrates and achieved an euthyroid state as well as normal cardiac enzyme levels without the use of antithyroid agents. Additionally, only a single case of recurrent angina in euthyroid state has been reported.…”

mentioning

confidence: 70%

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Acute Myocardial Infarction Associated With Painless Thyroiditis

Zhang

Shen

Dai

et al. 2014

The American Journal of the Medical Sciences

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confidence: 70%

“…4,5 A 26-year-old Chinese man was admitted to our department on an emergency basis because of a sudden onset of chest pain lasting for 2 hours. Thyrotoxicosis patients are susceptible to acute myocardial infarction (AMI), and especially if they have Grave's disease.…”

mentioning

confidence: 99%

Acute Myocardial Infarction Associated With Painless Thyroiditis

Zhang

Shen

Dai

et al. 2014

The American Journal of the Medical Sciences

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“…Coronary artery vasospasm is a recognized but rarely described cardiovascular manifestation of hyperthyroidism [6,7,8,9,10]. The primary mechanism of the malignant ventricular arrhythmias observed in this case was acute myocardial ischaemia during the occlusive vasospasm episodes.…”

Section: Case Reportmentioning

confidence: 80%

Amiodarone-Induced Destructive Thyroiditis Associated with Coronary Artery Vasospasm and Recurrent Ventricular Fibrillation

et al. 2012

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A 55-year-old male on long-term amiodarone therapy presented with ischaemic chest pain and recurrent unwitnessed syncope. Interrogation of his internal cardiac defibrillator, which had been inserted 4 years earlier, revealed two episodes of ventricular fibrillation, the timing of which corresponded to his syncopal events. Severe spontaneous coronary artery vasospasm was observed on coronary angiogram. Thyroid function testing revealed severe hyperthyroidism with a diagnosis of type 2 amiodarone-induced thyrotoxicosis (AIT) subsequently made. Treatment with prednisolone therapy was commenced and thyroid function rapidly normalized. Prednisolone was weaned without recurrence of hyperthyroidism and on last review, 6 months after initial presentation, he remains free from further chest pain and arrhythmias. Our patient's presentation is a very rare case of AIT-associated coronary artery spasm and documented ischaemic ventricular fibrillation with fortunate survival.

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“…Treatment of thyrotoxicosis-related coronary vasospasm consists of anti-thyroid and anti-anginal medications [1]. Data supports the use of nitrates and calcium channel blockers over beta-blockers due to the possibility of alpha-adrenergic stimulation and subsequent enhanced coronary vasoconstriction [10]. …”

Section: Discussionmentioning

confidence: 99%

Thyrotoxicosis Mimicking ST Elevation Myocardial Infarction

Marchena

Gutman

Zaidan

et al. 2017

Cureus

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Hyperthyroidism is well known to be associated with cardiac disease. Delay in making the diagnosis and occurrence of complications are common and are associated with a worse outcome.A 54-year-old male, non-smoker, with no past medical history and no significant family history presented to our hospital with severe left sided chest pain, “crushing” in nature. Electrocardiogram showed ST-segment elevations in the inferior leads. Troponin I level was 0.32 ng/mL (normal range 0-0.05 ng/mL) on presentation. The patient underwent an emergent coronary angiography which showed no evidence of occlusive coronary artery disease. The patient’s symptoms and signs prompted a high suspicion of thyrotoxicosis which was subsequently confirmed by a low thyroid stimulating hormone and high free thyroxine levels. The patient was given Methimazole and atenolol and his symptoms resolved.Awareness of coronary vasospasm due to thyrotoxicosis should be raised in patients presenting with typical angina pectoris with subsequent normal coronary angiographic results. History and physical examination may suggest underlying hyperthyroidism, but the absence of typical findings does not rule out the diagnosis.

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Thyrotoxicosis-Induced Acute Myocardial Infarction Due to Painless Thyroiditis

Abstract: Thyrotoxicosis due to painless thyroiditis provoked AMI in a young man who had no atherosclerotic coronary lesions and no CAD risk factors.

Cited by 18 publications

References 16 publications

Acute Myocardial Infarction Associated With Painless Thyroiditis

Acute Myocardial Infarction Associated With Painless Thyroiditis

Amiodarone-Induced Destructive Thyroiditis Associated with Coronary Artery Vasospasm and Recurrent Ventricular Fibrillation

Thyrotoxicosis Mimicking ST Elevation Myocardial Infarction

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