Mycotoxins in groundnuts, with special reference to aflatoxin

Keenan, J. I.; Savage, G. P.

doi:10.1007/978-94-011-0733-4_13

Cited by 4 publications

(3 citation statements)

References 110 publications

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“…Aflatoxin contamination can occur either before or after the crop is harvested. Preharvest contamination is actually the major economic problem in the peanut industry (3). One of the strategies to decrease the risk of peanut aflatoxin contamination is the use of genotypes with resistance to Aspergillus infection.…”

Section: Introductionmentioning

confidence: 99%

Aflatoxin Production in Six Peanut (Arachis hypogaea L.) Genotypes Infected with Aspergillus flavus and Aspergillus parasiticus, Isolated from Peanut Production Areas of Cordoba, Argentina

Ramón

Barrionuevo²,

Giorda³

et al. 2005

J. Agric. Food Chem.

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Aflatoxin contamination is one of the main factors affecting peanut seed quality. One of the strategies to decrease the risk of peanut aflatoxin contamination is the use of genotypes with resistance to Aspergillus infection. This laboratory study reports the resistance to Aspergillus infection and aflatoxin contamination of six peanut genotypes inoculated with 21 Aspergillus isolates obtained from the peanut production region of Cordoba, Argentina. The resistance was investigated in the seed coat and cotyledons of three resistant genotypes (J11, PI 337394, and PI 337409) and three breeding lines (Manfredi 68, Colorado Irradiado, and Florman INTA) developed at the Instituto Nacional de Tecnologia Agropecuaria (INTA), Manfredi Experimental Station, Cordoba, Argentina. Resistance to fungal colonization and aflatoxin contamination was found to be associated with seed coat integrity in the PI 337394, PI 337409, and J11 genotypes, whereas the INTA breeding lines such as Colorado Irradiado showed a moderate resistance and the Manfredi 68 and Florman INTA genotypes the least resistance. Furthermore, another type of resistance associated with cotyledons was found only in the PI 337394 genotype.

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Section: Introductionmentioning

confidence: 99%

Aflatoxin Production in Six Peanut (Arachis hypogaea L.) Genotypes Infected with Aspergillus flavus and Aspergillus parasiticus, Isolated from Peanut Production Areas of Cordoba, Argentina

Ramón

Barrionuevo²,

Giorda³

et al. 2005

J. Agric. Food Chem.

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“…Aflatoxins can accumulate during food storage, particularly under hot, humid conditions, where there is additional risk of rodent and insect damage. Food spoilage and contamination with mycotoxins can be a significant problem in circumstances of traditional storage at the local farm level 56,57 . Efforts to remove aflatoxins post harvest by some form of processing have been mainly pursued as a commercial activity, and are appropriate only for feeds, as they alter organoleptic properties of foods, making them unfit for human consumption 58,59 …”

Section: Primary Prevention Of Hepatocellular Carcinomamentioning

confidence: 99%

“…Food spoilage and contamination with mycotoxins can be a significant problem in circumstances of traditional storage at the local farm level. 56,57 Efforts to remove aflatoxins post harvest by some form of processing have been mainly pursued as a commercial activity, and are appropriate only for feeds, as they alter organoleptic properties of foods, making them unfit for human consumption. 58,59 In terms of post-harvest control at the subsistence farm level, the main aims are to minimize fungal growth and aflatoxin production.…”

Section: Post-harvest Aflatoxin Interventionmentioning

confidence: 99%

The role of aflatoxins and hepatitis viruses in the etiopathogenesis of hepatocellular carcinoma: A basis for primary prevention in Guinea–Conakry, West Africa

Turner

Sylla

Diallo

et al. 2002

J of Gastro and Hepatol

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Aflatoxins and hepatitis B virus (HBV) are major risk factors for hepatocellular carcinoma (HCC) in South-east Asia and Africa, parts of the world where this cancer is most prevalent. Exposure to both factors is endemic, occurring from early in life. There is evidence from both epidemiological studies and animal models that the two factors can act synergistically to increase the risk of HCC, but the underlying cellular and molecular mechanisms of interaction are as yet undefined. One possibility suggested by studies in HBV transgenic mice is that chronic liver injury alters the expression of carcinogen metabolizing enzymes, thus modulating the level of binding of aflatoxin to DNA. Primary prevention of HCC in high incidence areas of the world should primarily be focused on provision of the safe, effective vaccine against HBV. However, measures to reduce the high levels of aflatoxin exposure, where chronic HBV infection is currently epidemic, would also significantly contribute to reducing HCC incidence. In Guinea-Conakry, West Africa, surveys of HBV infection and aflatoxin exposure have established baseline data for the implementation of a community-based intervention study. This study will evaluate the effectiveness of improving the post-harvest processing and storage of the groundnut crop, a major source of aflatoxins, using aflatoxin-albumin adducts as the outcome measurement.

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