“…Genes involved in signal transduction ( STAT6 , Table IV), malignant transformation ( TP53 , P21 , KRAS , Table IV; BRAF ), apoptosis ( FAS , Table I; FASLG , Table I) and galactose metabolism ( GAL T ,Table IV) have been investigated for a role in endometriosis susceptibility, but no consistent association has been found (Tables I–IV) (Cramer et al ., 1996; Morland et al ., 1998; Hadfield et al ., 1999; Hsieh et al ., 2001c; Stefansson et al ., 2001; Chang et al ., 2002; Lattuada et al ., 2004b; Omori et al ., 2004; Fernandez et al ., 2005; He et al ., 2006; Hsieh and Lin, 2006; Zhao et al ., 2006; Bhanoori et al ., 2007; Vietri et al ., 2007). Two exceptions are the STAT6 3′-UTR 2964G/A polymorphism (Bhanoori et al ., 2007) and the GALT p.N314D polymorphism (Cramer et al ., 1996), which are over-represented among South Indian women and US women with endometriosis, respectively.…”