“…[1][2][3]9,[51][52][53][54][55][56]59,64,71,73,76,86,[88][89][90][91][92][93] As in the recipient, old age, pre-existing hypertension, diabetes mellitus, subclinical kidney disease, and nephron under dosing (innate, female to male donor, old age, and donorrecipient body mass index mismatch) represent potential causes for development or aggravation of hypertension posttransplant. [1][2][3]9,59,64,71,73,[91][92] New evidence is emerging regarding the potential role of the donor genetic makeup in the pathogenesis of posttransplant hypertension.…”