Moniliasis and Moniliids

Hopkins, J. G.

doi:10.1001/archderm.1932.01450020619001

Cited by 48 publications

(5 citation statements)

References 10 publications

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“…If one is willing to allow the existence of hidden foci of Candida in the bowel, as Hopkins has done, it becomes all too easy to regard any type of skin lesion as a moniliid. 28 Moniliids have never been observed in our clinical or experimental experience. In conclusion, the sum of our experience does not add up to proof of the hypersensitivity state in moniliasis.…”

Section: Susceptibility Of Various Regionsmentioning

confidence: 77%

The Biology of Experimental Human Cutaneous Moniliasis (Candida Albicans)

Maibach¹

1962

Arch Dermatol

166

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Section: Susceptibility Of Various Regionsmentioning

confidence: 77%

The Biology of Experimental Human Cutaneous Moniliasis (Candida Albicans)

Maibach¹

1962

Arch Dermatol

166

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“…The frequent occurrence of candida infections in patients with diabetes mellitus has been recognised for many years (Plass et al, 1931;Hopkins, 1932;Akrawi, 1960;Winner and Hurley, 1964;Hesseltine, 1965). The predisposing effect of diabetes has been confirmed by animal experiments (Andriole and Hasenclever, 1962;Hurley, 1966;Louria, et al, 1966;Van Cutsem and Thienpont, 1971) in which an induced diabetic state was associated with increased susceptibility to infection by pathogenic Candida species.…”

mentioning

confidence: 99%

Prevalence of pathogenic yeasts and humoral antibodies to candida in diabetic patients.

Odds¹,

Evans²,

Taylor³

et al. 1978

J Clin Pathol

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SUMMARY The prevalence of oral yeasts and humoral precipitating antibodies to candida was estimated in 204 unselected diabetic patients (172 outpatients and 32 inpatients). Yeasts, mainly Candida albicans, were isolated from the mouths of 41 % of the outpatients and precipitins were found in 17-5 % although none of the patients had clinically overt candidiasis. The extent of oral yeast colonisation and incidence of antibodies was not related to their antidiabetic treatment or to the duration of their diabetes. It was, however, related to the blood glucose and urine sugar levels at the time they were sampled, the highest incidence being among the diabetic inpatients with high blood glucose levels at the time of sampling and the lowest among outpatients with normal blood glucose levels at the time of sampling. There was no such correlation when diabetic control over the previous 12-month period was considered.

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“…Other investigators, however, believed that the disease was caused by infection of the gastrointestinal tract (8), and Dillaha et al (9) treated an AE patient with diiodohydroxyquinoline for a yeast infection. The drug controlled the symptoms of AE and subsequently became the treatment of choice although its mechanism of action is still unknown (4, 10, 11).…”

mentioning

confidence: 99%