Mixed anaerobic periapical infection with sinus tract

Haapasalo, Markus; Ranta, Klaus; Ranta, Helena

doi:10.1111/j.1600-9657.1987.tb00548.x

Cited by 31 publications

(20 citation statements)

References 14 publications

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“…Introduction of interim systemic antibiotic therapy may be indicated in some cases of persisting periapical lesions; however, since the body of the lesion is rarely heavily contaminated with bacteria, antimicrobial therapy often fails to promote healing ( 48, 87, 114).…”

Section: Conclusion – Implications For Therapymentioning

confidence: 99%

Protective and destructive immune reactions in apical periodontitis

Márton¹,

Kiss

2000

Oral Microbiology and Immunology

207

193

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Apical periodontitis is initiated primarily by the mixed microflora of infected root canals. Continuous flow of bacteria and their products through the apical foramen induces influx, activation and coordinated interaction of immune-inflammatory cells within the periapical area. Successful mobilization of host defense mechanisms prevents abundant extraradicular bacterial invasion. However, anti-infective effector mechanisms are not restricted to killing the invading microorganisms but also destroy normal tissue components and induce bone absorption, resulting ultimately in the loss of the affected teeth. Moreover, autocrine and paracrine loops of stimulation may lead to the perpetuation of the local inflammatory lesion and may also alter the function of remote tissues and organs. This review attempts to summarize current knowledge about the pathogenic mechanism of apical periodontitis, focusing on the formation of a special granulation tissue that effectively fights bacteria originated from the infected pulp chamber and, by exerting this protective function, also contributes to harmful local and distant events. The dynamic equilibrium between defensive and destructive mechanisms may provide a pathobiological basis for better understanding of clinical signs and symptoms of various forms of apical periodontitis lesions and influence treatment strategy and practice.

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Section: Conclusion – Implications For Therapymentioning

confidence: 99%

Protective and destructive immune reactions in apical periodontitis

Márton¹,

Kiss

2000

Oral Microbiology and Immunology

207

193

View full text Add to dashboard Cite

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“…Microbiological culture of the apical granulomatous tissue yielded Actinomyces israelii infection. Haapasalo et al . (1987) showed the failure of metronidazole therapy in cases with Bacteroides intermedius‐ associated anaerobic infection.…”

Section: Discussionmentioning

confidence: 99%

Effect of occlusal trauma on healing of periapical pathoses: report of two cases

Harn

Chen²,

Chen

et al. 2001

Int Endodontic J

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Occlusal trauma is positively correlated with changes in periodontal tissues. Although experiments in animals have shown that application of forces to teeth will not induce further periodontal destruction, we believe that some failures of root canal treatment may be due to the presence of occlusal trauma modulating the responses of inflamed periapical tissues or apical pathoses with persistent infection.

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“…Intraradicular bacterial and fungal species that might cause secondary periapical lesions are Streptococcus, Propionibacterium , and Candida (especially Candida albicans ). It is often difficult to identify the etiology of periradicular lesions, because the bacteria might also be extraradicular, spreading to the periapical area . This is confirmed by the fact that extraradicular bacteria are frequently found in the presence of an abscess or fistula, which are clinical symptoms of secondary periapical lesions .…”

Section: Microbiology Of Odontogenic Sinusitismentioning

confidence: 99%

Pathophysiology of sinusitis of odontogenic origin

Taschieri

Torretta

Corbella

et al. 2015

J of Invest & Clin Dent

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Sinusitis of odontogenic origin, which is frequently encountered in routine otolaryngological and dental clinical practice, has been described as a reactive maxillary inflammation secondary to maxillary tooth infection or trauma to an odontogenic disease of maxillary bone, dental extractions, implant placement, or endodontic treatment impairing the integrity of the Schneiderian membrane. The aim of the present review was to investigate and discuss the most recent pathophysiological findings, predisposing odontogenic factors, microbiology, and the possible involvement of bacterial biofilms (BB) in the development of sinusitis. The narrative literature review showed that there might be a correlation between the bacteria present in pathological teeth in communication with the sinus and those found in infected sinus. The formation of a BB might be also involved in the etiopathogenesis of sinusitis of odontogenic origin. In conclusion, the true origin of odontogenic sinusitis is still unresolved. In clinical terms, the choice of suitable therapy depends on the characteristics of the biofilm. Further microbiological studies are required to better investigate the role of BB.

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Mixed anaerobic periapical infection with sinus tract

Cited by 31 publications

References 14 publications

Protective and destructive immune reactions in apical periodontitis

Protective and destructive immune reactions in apical periodontitis

Effect of occlusal trauma on healing of periapical pathoses: report of two cases

Pathophysiology of sinusitis of odontogenic origin

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