A 71-year-old patient presented with a 3-year history of paroxysmal, rapid regular palpitations despite trials of sotalol and diltiazem. A previous Holter study had documented a narrow QRS tachycardia (Fig. 1). The patient underwent an electrophysiology study (EPS) during which a tachycardia was inducible with programmed atrial stimulation (Fig. 2). What is the diagnosis?The Holter rhythm strips A and B (Fig. 1) document a narrow QRS tachycardia. The tachycardia is initiated by an atrial premature beat (APB) (third beat in Panel I) without a P wave to R wave interval (PR) jump. After the first four cycles, atrial but not ventricular activity becomes regular. The irregularity in RR cycle length (CL) persists until the end of Panel II, at which time it too becomes regular. The CL of the tachycardia in Panel III is 340 msec (shorter than in Panel II by 20 msec). The relationship between the ventricular and atrial excitation is not constant in Panels I and II. In Panel III, there is a constant and short RP relationship favoring atrioventricular nodal reentrant tachycardia (AVNRT). The irregularity in the RP relationship in Panels I and II could potentially be explained by AVNRT with variable block to the ventricle. An alternative explanation would be an atrial Figure 1. Holter strips A and B showing initiation of tachycardia in Panel I.tachycardia initiated by an APB without a PR jump exhibiting variable conduction to the ventricles initially (Panels I and II) but later stabilizing to 1:1 conduction (Panel III). Analyzing Figure 1 further reveals a change in the morphology of the QRS, clearly seen in the "B" rhythm strip, in the first two as well as the last three complexes of Panel II and throughout Panel III. This raises the suspicion of two tachycardias, the initiation being an atrial tachycardia (Panel I) that transforms by the end of Panel II into an AVNRT. The tachycardia in Panels I and II cannot be AVRT because of the varying R and P relationship. In Panel III, the RP relationship becomes constant, but the short RP interval makes a diagnosis of AVRT unlikely.At EPS, the arrhythmia induced is also a narrow QRS tachycardia at CL of 315 msec (Fig. 2). Though there is no significant change in the CL, from the fourth QRS complex onward subtle changes in QRS morphology are apparent. The intracardiac recording of Figure 3 reveals that the first three beats have the earliest local activation in the high right atrium, suggesting the mechanism to be atrial tachycardia (the short Figure 2. 12-Lead surface ECG of tachycardia initiated at EPS.