Maternal and anthropomorphic risk factors for shoulder dystocia

Mazouni, Chafika; Porcu, G.; Cohen‐Solal, Emmanuelle; Heckenroth, H.; Guidicelli, B.; Bonnier, Pascal; Gamerre, M.

doi:10.1080/00016340600605044

Cited by 62 publications

(35 citation statements)

References 21 publications

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“…Mazouni et al stated that obesity is a significant maternal risk factor for shoulder dystocia, but also specified that the risk is caused by discrepancy between maternal height or weight and infant weight [22]. In contrast, Robinson et al found that obesity cannot be seen as an independent risk factor and they stated that fetal macrosomia is the single most powerful predictor in their study [23].…”

Section: Discussionmentioning

confidence: 94%

Maternal and perinatal outcome in obese pregnant patients

Schrauwers

Dekker

2009

The Journal of Maternal-Fetal & Neonatal Medicine

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Section: Discussionmentioning

confidence: 94%

Maternal and perinatal outcome in obese pregnant patients

Schrauwers

Dekker

2009

The Journal of Maternal-Fetal & Neonatal Medicine

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“…Consequently, persistent brachial palsy with irreversible changes found in about 3% of patients one year after birth, with recovery in upper brachial palsy possible in about 80% (Duchen-Erb) and in lower in 40% of cases (Klumpke). About 40% of SD appears in children with birth weight below 4000g, although fetal macrosomia in babies with birth weight above 4000-4500g is also a predicting factor for development of SD, usually because of relative fetopelvine disproportion [9,10]. Injuries in newborns tend to develop as a consequence of any of the following two conditions -either protracted shoulder incarceration (hypoxia/ acidosis), or complications as a consequence of hand maneuvers (uncritical head and neck tractionspinal cord injury without radiographic abnormalities /SCIWORA/, brachial palsy, injuries of neck organs).…”

Section: Discussionmentioning

confidence: 99%

Refractory shoulder dystocia in term macrosomic newborn with verified complete meningoencephalomyelocele

Habek¹,

Horvat²,

Kulaš³

et al. 2008

Open Medicine

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AbstractThe authors presented the birth course, in a primiparous woman with complicated, refractory, high shoulder dystocia, of a macrosomic term-born malformed child (4400/54) with complete occipital meningoencephalomyelocele and microcephaly. After head delivery, high shoulder dystocia occurred that could not be resolved with neither the McRoberts or Resnik maneuver nor with the Woods and Barnum maneuvers, despite recurred tries. Only on third attempt with the Barnum maneuver was the posterior arm released with hand traction, followed by the whole body of the macrosomic baby. The child was transferred to the Neurosurgery department where operative correction was performed, and after the intervention the child died because of respiratory insufficiency. Due to the strict parental decision on pregnancy continuation and prohibition of any obstetric interventions during delivery, unborn child and parturient, as well as the obstetric team, were put into high professional, forensic and ethical risk.

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“…This finding implies that inadequate screening for glucose intolerance and superficial treatment of diabetes are conducive to these injuries. This being the case, implementation of the proposed measures could probably reduce the rate of shoulder dystocia markedly both in America and in other countries, where its rate has also increased in recent years [1,9,[33][34][35][36]. The "CESDI Committee", having investigated shoulder dystocia related fetal deaths in England, Wales and Northern Ireland, found substandard intrapartum management the most frequent causative factor [3].…”

Section: Discussionmentioning

confidence: 99%

Shoulder dystocia related fetal neurological injuries: the role of diabetic control

Iffy¹,

Brimacombe

Váradi³

et al. 2009

Open Medicine

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AbstractThe study explores the roles of routine prenatal diabetic screening and control in the occurrence of neurological birth injuries associated with shoulder dystocia. The investigation involved retrospective review of 226 medical records that contained information about the antenatal events in cases that resulted in permanent neonatal injuries following arrest of the shoulders at delivery. Close attention was paid to diabetic screening and management of mothers with evidence of glucose intolerance. Analysis of the records revealed that one-third of all women, including those with predisposing factors, received no diabetic screening during pregnancy. The majority of confirmed diabetic patients were not treated adequately. Among babies of diabetic women, birth weights exceeding 4500 g were about 30-fold more frequent than among those with normal glucose tolerance. The data suggest that universal screening and rigid diabetic control, including mothers with borderline glucose tolerance, are effective measures for the prevention of excessive fetal growth and intrapartum complications deriving from it. If ignored, impaired maternal glucose tolerance may become a major predisposing factor for neurological birth injuries. It appears therefore that with routine screening for diabetic predisposition and effective control of gestational diabetes the risk of fetal damage can be reduced substantially.

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Maternal and anthropomorphic risk factors for shoulder dystocia

Abstract: Shoulder dystocia may be anticipated in cases involving short women and discrepancy between maternal height or weight and infant weight.

Cited by 62 publications

References 21 publications

Maternal and perinatal outcome in obese pregnant patients

Maternal and perinatal outcome in obese pregnant patients

Refractory shoulder dystocia in term macrosomic newborn with verified complete meningoencephalomyelocele

Shoulder dystocia related fetal neurological injuries: the role of diabetic control

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