Marshall's syndrome

Fontenelle, Elisa; Almeida, Ana Paula Moura de; Souza, Gabriela Maria Assis de Almeida

doi:10.1590/s0365-05962013000200020

Cited by 12 publications

(2 citation statements)

References 10 publications

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“…Acquired cutis laxa type II presents as crops of inflammatory erythematous papules and plaques associated with constitutional symptoms. The inflammatory skin lesions may be triggered by vaccinations, drugs (penicillin, penicillamine, isoniazid), arthropod bites, or diseases like Sweet syndrome, syphilis, and systemic lupus erythematosus and may last from weeks to months resolving with localized or generalized cutis laxa 1,2,4,5 . Histopathologically, the inflammatory phase reveals dermal neutrophilic infiltrate with capillary dilatation and dermal edema.…”

Section: Discussionmentioning

confidence: 99%

“…The inflammatory skin lesions may be triggered by vaccinations, drugs (penicillin, penicillamine, isoniazid), arthropod bites, or diseases like Sweet syndrome, syphilis, and systemic lupus erythematosus and may last from weeks to months resolving with localized or generalized cutis laxa. 1,2,4,5 Histopathologically, the inflammatory phase reveals dermal neutrophilic infiltrate with capillary dilatation and dermal edema. The elastic fibers become short, fragmented, or decreased in number in late stages, but this may be difficult to appreciate without a control biopsy for comparison.…”

Section: Discussionmentioning

confidence: 99%

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Acquired cutis laxa type II (Marshall syndrome) in a 3‐month‐old boy

Sindhuja

Narayanan

Gupta

et al. 2021

Pediatric Dermatology

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Acquired cutis laxa type II (Marshall syndrome) is a post‐inflammatory elastolysis occurring in infancy and childhood. It is challenging to treat with very few effective treatment options available. Herein, we describe the case of a 3‐month‐old boy with acquired cutis laxa type II secondary to a neutrophilic dermatosis. Early treatment of the initial inflammatory phase is essential to reduce the permanent sequelae.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Acquired cutis laxa type II (Marshall syndrome) in a 3‐month‐old boy

Sindhuja

Narayanan

Gupta

et al. 2021

Pediatric Dermatology

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Acquired Disorders of Dermal Connective Tissue

Lovell

2016

Rook's Textbook of Dermatology, Ninth Edition

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The subject matter of this chapter comprises the major acquired disorders that affect mainly collagen and elastic tissue. The connective tissue of normal skin is discussed elsewhere. Heritable disorders of connective tissue, such as Ehlers–Danlos syndrome, are covered in another chapter, as are disorders of ground substance, such as the mucinoses and mucopolysaccharidoses. In addition, many disorders, including developmental defects, neoplasms and other reactive or scarring conditions, may involve the connective tissues. Although it can be convenient to separate conditions into disorders affecting collagen or elastic tissue, the distinction is somewhat artificial, as changes in these proteins are generally preceded by abnormal metabolism of other matrix components, such as fibronectin or periostin.

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Acquired Disorders of Dermal Connective Tissue

Fahy,

Lovell

2024

Rook's Textbook of Dermatology

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This chapter reviews acquired changes in dermal connective tissue during normal ageing and in disease processes, including photodamage. Corticosteroids are a potent cause of dermal atrophy, which may also be congenital or induced by inflammatory disorders related to morphoea. Enzymatic degradation of elastic tissue results in localised or generalised skin laxity, which resembles genetic disorders such as pseudoxanthoma elasticum and cutis laxa. In contrast, some disorders are characterised by excessive deposition of collagen or elastic tissue. Fibrosis is associated with contracture, which causes deformity and restricted movement as in palmoplantar fibromatosis. Several chemical environmental triggers induce diffuse fibrosis resembling systemic sclerosis. Keloids represent an abnormal scarring response to trauma. Finally, dermal connective tissue components may be extruded via the epidermis in perforating disorders such as elastosis perforans serpiginosa.

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Marshall's syndrome

Cited by 12 publications

References 10 publications

Acquired cutis laxa type II (Marshall syndrome) in a 3‐month‐old boy

Acquired cutis laxa type II (Marshall syndrome) in a 3‐month‐old boy

Acquired Disorders of Dermal Connective Tissue

Acquired Disorders of Dermal Connective Tissue

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