Mandibular coronoid process locking: A prospective study of frequency and association with internal derangement of the temporomandibular joint

Isberg, Annika; Isacsson, Göran; Nah, Kyung-Soo

doi:10.1016/0030-4220(87)90189-7

Cited by 102 publications

(41 citation statements)

References 24 publications

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“…Some authors believe coronoid hyperplasia could be reactive to internal derangement of the TMJ which does not hold well in our case [4][5][6]. Genetic inheritance and syndromic associations have also been suggested [6,7].…”

Section: Discussioncontrasting

confidence: 60%

Congenital Hypertonia of the Temporalis Leading to Trismus Since Birth

Raja¹,

Jones²,

et al. 2016

J. Maxillofac. Oral Surg.

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Section: Discussioncontrasting

confidence: 60%

Congenital Hypertonia of the Temporalis Leading to Trismus Since Birth

Raja¹,

Jones²,

et al. 2016

J. Maxillofac. Oral Surg.

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“…Various theories have been proposed regarding the etiology of coronoid hyperplasia, including trauma, hyperactivity of the temporalis muscle, dysfunction of the TMJ caused by chronic disc displacement, endocrine stimuli and genetic and familial factors [2,8,9]. It has been hypothesized that the enlarging osteochondroma compresses against the zygoma and either resorbs or remodels it causing a pseudo joint [4].…”

Section: Discussionmentioning

confidence: 99%

Imaging and Histopathological Features of Jacob’s Disease: A Case Study

Ajila

Hegde

Gopakumar³

et al. 2011

Head and Neck Pathol

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“…e710 Discussion Jacob's disease is an uncommon condition, with few reports in the literature. The proposed causes include temporal muscle hyperactivity, which could lead to increased vascular supply and reactive elongation of the coronoid process (2). Other proposed causes are previous trauma (as in our patient), chronic disc displacement, endocrine disruptions, and genetic abnormalities (3)(4)(5)(6)(7).…”

Section: Case Descriptionmentioning

confidence: 78%

Jacob s disease secondary to coronoid process osteochondroma. A case report

Coll-Anglada¹,

Acero²,

Vila-Masana³

et al. 2011

Med Oral

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The formation of a new joint between a pathologically elongated coronoid process and the body of the malar homolateral bone is known as Jacob's disease. Coronoid process hyperplasia was first described in 1853 by Von Langenbeck, and it was not until 1899 when Oscar Jacob described the disease that it was named after him. Jacobs's disease is an uncommon entity with only a few documented cases in the literature. The condition first manifests with progressive limitation of mouth opening and facial asymmetry. Pain is uncommon and it mainly affects young patients. Different factors have been postulated as possible causes, including temporal muscle hyperactivity, previous trauma, chronic disc displacement of the ipsilateral temporomandibular joint, endocrine stimuli, and genetic alterations. Definitive diagnosis is by histopathology and it is necessary to confirm bone hyperplasia, the presence of cartilage and synovial capsule forming the new joint between the malar bone and the coronoid process. We report a 52-year-old woman patient with a history of childhood trauma in the right preauricular region. She came to our department with a 2-year history of progressive limitation of mouth opening. Computed tomography (CT) revealed a right coronoid process elongation, in contact with the homolateral malar bone, causing it to deform. Surgery with general anesthesia was performed using an intraoral vestibular approach. Histopathology confirmed the diagnoses of Jacob's disease.

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Mandibular coronoid process locking: A prospective study of frequency and association with internal derangement of the temporomandibular joint

Cited by 102 publications

References 24 publications

Congenital Hypertonia of the Temporalis Leading to Trismus Since Birth

Congenital Hypertonia of the Temporalis Leading to Trismus Since Birth

Imaging and Histopathological Features of Jacob’s Disease: A Case Study

Jacob s disease secondary to coronoid process osteochondroma. A case report

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