Longitudinal study of height and weight at adolescence.

Buckler, John; Wild, J. Martin

doi:10.1136/adc.62.12.1224

Cited by 48 publications

(41 citation statements)

References 11 publications

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“…There are likely to be bigger differences in body size (weight and height) between the younger and older boys than between the younger and older girls (Buckler & Wild, 1987). Finally, we found a significant main effect for sex for distance from the wall which post hoc comparisons indicated that distance was greater for boys than girls.…”

Section: Competition In 229 M Of Watersupporting

confidence: 54%

Racing Start Safety: Head Depth and Head Speed During Competitive Swim Starts into a Water Depth of 2.29 m

Cornett¹,

White²,

Wright³

et al. 2011

IJARE

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The head depths and head speeds of swimmers attained following the execution of racing starts during competition have not been well described. To address this, 211 competitive starts were filmed into a starting depth of 2.29 m with a block height of 0.76 m. Starts were stratified according to age, sex, stroke, and swim meet. Dependent measures were maximum depth of the center of the head, head speed at maximum head depth, and distance from the wall at maximum head depth. Significant main effects existed for age for all three measures: F(1, 106) = 13.33, p < .001, F(1, 106) = 18.60, p < .001 and F(1, 106) = 70.59, p < .001, respectively. There was a significant age by sex interaction, F(1, 106) = 5.36, p = 0.023, for head speed. In conclusion, older swimmers performed starts that were deeper and faster than younger swimmers and nearly all starts exceeded the threshold speeds for injury. As compared to starts previously reported into 1.22 m, starts were deeper, slower, and farther from the starting wall at maximum head depth.

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Section: Competition In 229 M Of Watersupporting

confidence: 54%

Racing Start Safety: Head Depth and Head Speed During Competitive Swim Starts into a Water Depth of 2.29 m

Cornett¹,

White²,

Wright³

et al. 2011

IJARE

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show abstract

“…In the studies by Marshall and Tanner [16,17] based on clinical criteria (as in this study) the average age of onset of puberty was 11.6 years in boys (n = 288) and 11.2 years in girls (n = 192), while TPG was 29.5 and 19.2 cm in boys and girls, respectively. In the British cohort investigated by Buckler and Wild [19] the age at puberty onset was 12.2 years with a TPG of 28.0 cm in boys (n = 128) and 11.1 years with a TPG of 21.0 cm in girls (n = 83), respectively. Ethnicity and secular developments may also impact on the timing and extent of pubertal growth [21,22].…”

Section: Discussionmentioning

confidence: 99%

“…Even in studies on growth and development in normal children, depending on the method used – visible appearance of pubertal stages [16,17,18,19] or the nadir of height velocity based on longitudinal growth analysis [20] – different time points of puberty onset and differences of the observed TPG were established. In the studies by Marshall and Tanner [16,17] based on clinical criteria (as in this study) the average age of onset of puberty was 11.6 years in boys (n = 288) and 11.2 years in girls (n = 192), while TPG was 29.5 and 19.2 cm in boys and girls, respectively.…”

Section: Discussionmentioning

confidence: 99%

Observed and Predicted Total Pubertal Growth during Treatment with Growth Hormone in Adolescents with Idiopathic Growth Hormone Deficiency, Turner Syndrome, Short Stature, Born Small for Gestational Age and Idiopathic Short Stature: KIGS Analysis and Review

Ranke

Lindberg²

2011

Horm Res Paediatr

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Background/Aims: The aim was to describe the factors determining total pubertal growth (TPG) in adolescents with growth hormone deficiency (GHD), Turner syndrome (TS), who were small for gestational age (SGA) or had idiopathic short stature (ISS). Methods: The patients were documented within KIGS (Pfizer International Growth Database). TPG was defined as growth from puberty onset – spontaneous (>B1 or testes >3 ml) or induced with sex steroids to adult height in idiopathic GHD (n = 639; males 399), TS (n = 463), SGA (n = 59; males 35), and ISS (n = 130; males 87). Algorithms to predict TPG at puberty onset were developed by multiple linear regression analysis. Results: In GHD (males, females) and TS TPG could be explained with the same four predictors: (1) age (years) at puberty onset (neg.), (2) age – bone age at puberty onset (pos.), (3) height – midparental height (SDS) at puberty onset (neg.), and (4) mean dose of growth hormone (GH) during puberty (pos.). The algorithms explained 66, 65 and 68%, respectively, of the variability with total errors of 4.5, 3.8 and 2.9 cm, in spontaneous and induced TPG. Conclusions: TPG is determined by the same factors at puberty onset in adolescents with GHD, TS, SGA or ISS treated with GH. Thus, TPG depends mostly on the outcomes achieved at the end of prepubertal growth and less so on the dose of GH.

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“…Although a deceleration of growth velocity before puberty is physiological, no significant change in height SDS before puberty is expected in normal children [16]. Growth during puberty, expressed as total height gain and growth velocity, was reduced when compared to normal subjects [12, 13], regardless of hypogonadism. Final height SDS was not significantly different from height SDS for CA at the beginning of puberty in patients with spontaneous puberty, while it was even improved in patients with induced puberty.…”

Section: Discussionmentioning

confidence: 99%

“…PHV was the highest growth velocity observed during a 6- to 12-month period from the start of puberty to final height. Buckler standards were used to evaluate PHV and PHG [12, 13]. …”

Section: Methodsmentioning

confidence: 99%

No Difference in Pubertal Growth and Final Height between Treated Hypogonadal and Non-Hypogonadal Thalassemic Patients

et al. 2004

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Background: Many factors can negatively affect growth in thalassemic patients, and hypogonadism has been considered as the main factor responsible for their pubertal growth failure. Objective: To evaluate the influence of hypogonadism and its treatment on pubertal growth and final height in thalassemic patients. Methods: We compared the growth of 28 hypogonadal thalassemic patients in whom puberty was induced to that of 25 patients in whom puberty occurred spontaneously. Results: In both groups of patients we observed reduced peak height velocity (induced puberty: females 4.9 ± 2.1, males 6.0 ± 1.8 cm/year; spontaneous puberty: females 6.1 ± 1.5, males 7.3 ± 2.1 cm/year) and pubertal height gain (induced puberty: females 11.3 ± 4.0, males 18.0 ± 4.5 cm/year; spontaneous puberty: females 15.8 ± 2.7, males 18.1 ± 5.3 cm/year) and a short final height (induced puberty: females –1.8 ± 0.7, males –2.1 ± 1.0 SDS; spontaneous puberty: females –2.3 ± 1.0, males –1.9 ± 1.0 SDS). Conclusions: Poor pubertal growth is present in thalassemic patients regardless of hypogonadism. Other factors are responsible for the reduced growth spurt and the final short stature observed in these patients.

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Longitudinal study of height and weight at adolescence.

Cited by 48 publications

References 11 publications

Racing Start Safety: Head Depth and Head Speed During Competitive Swim Starts into a Water Depth of 2.29 m

Racing Start Safety: Head Depth and Head Speed During Competitive Swim Starts into a Water Depth of 2.29 m

Observed and Predicted Total Pubertal Growth during Treatment with Growth Hormone in Adolescents with Idiopathic Growth Hormone Deficiency, Turner Syndrome, Short Stature, Born Small for Gestational Age and Idiopathic Short Stature: KIGS Analysis and Review

No Difference in Pubertal Growth and Final Height between Treated Hypogonadal and Non-Hypogonadal Thalassemic Patients

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