Linear fixed drug eruption

Sigal-Nahum, M; Konqui, Alain; Gaulier, A; Sigal, S

doi:10.1111/j.1365-2133.1988.tb02604.x

Cited by 42 publications

(22 citation statements)

References 4 publications

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“…Besides, our survey of the current literature has indicated that linear FDE with unilateral location is extremely rare. In 2003, Li et al [2] reported an FDE case with unilateral location in the right breast due to naproxen use; in 1987, Sigal-Nahum et al [15] reported a case of linear FDE on the left leg, and in 1997 Ozkaya-Bayazit and Baykal [16] reported a case of linear FDE induced by trimethoprim. We have found no case of calcium-acetate-related FDE in the literature.…”

Section: Case Reportmentioning

confidence: 99%

Calcium Acetate-Induced Linear Fixed Drug Eruption

Coşkun¹,

Saral²,

Öztürk³

et al. 2005

Dermatology

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Section: Case Reportmentioning

confidence: 99%

Calcium Acetate-Induced Linear Fixed Drug Eruption

Coşkun¹,

Saral²,

Öztürk³

et al. 2005

Dermatology

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“…Some acquired linear disorders such as lichen striatus [20], linear lichen planus [23], linear psoriasis [211 or linear fixed drug eruption [35] do not represent true nevi because they are not long-standing and because they appear in a mosaic area of skin that otherwise would have remained invisible.…”

Section: Distinction Between Nevoid Disorders and Nevimentioning

confidence: 99%

What is a Nevus

Happle

1995

Dermatology

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From the study of standard textbooks of dermatology, no comprehensive understanding of the word nevus can be derived. The purpose of this article is to formulate a workable definition. As a first step, various definitions proposed by previous authors are reviewed. Their inconsistency appears to be mainly due to the fact that the term nevus is used to denote both neoplastic and nonneoplastic, congenital and acquired as well as hereditary and nonhereditary skin lesions. Taking this difficulty into account, the author suggests, as a unifying concept, the new category of genetic mosaicism. The following definition is proposed: ‘Nevi are visible, circumscribed, long-lasting lesions of the skin or the neighboring mucosa, reflecting genetic mosaicism. With the exception of melanocytic nevi, they do not show neoplastic growth. They never show malignant neoplasia.’ -For several types of nevi the concept of genetic mosaicism has already been confirmed at the cellular level. Future cytogenetic and molecular studies should show whether the proposed definition is valid for all other types of nevi and thus generally acceptable.

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“…Lesions distributed along Blaschko lines may occur in patients with scattered lesions of lichen planus, but isolated blaschkolinear lichen planus, especially of the lower limbs, is also seen. Other conditions among the acquired inflammatory dermatoses that follow the lines of Blaschko and exhibit a histological lichenoid pattern are linear lichen nitidus [Prigent et al, 1989], the linear form of chronic graft versus host reaction (GVHR) [Wilson and Lockman, 1994], linear fixed drug eruption [Sigal-Nahum et al, 1988], and linear lupus erythematosus [Heid et al, 1996]. In all of these conditions, the GVHR, the intake of drugs, or the autoimmune antibodies are hypothesized to unmask tolerance of an abnormal keratinocyte clone that remained hidden in some embryonic Blaschko lines.…”

Section: Inflammatory Acquired Blaschkolinear Dermatosesmentioning

confidence: 99%

Acquired Blaschkolinear dermatoses

Grosshans

1999

Am. J. Med. Genet.

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Congenital and/or nevoid skin disorders following the lines of Blaschko may have a delayed onset after birth. They have to be differentiated from acquired dermatoses exhibiting the same linear pattern. In common dermatoses, such as psoriasis or lichen planus, lesions in a blaschkolinear distribution most often occur together with scattered lesions, but occasionally they may be isolated. Less common self-limited dermatoses such as lichen striatus and adult blaschkitis always present in a blaschkolinear fashion. In these diseases, or some other conditions occasionally distributed along these lines (chronic graft versus host reaction, fixed drug eruption, lupus erythematosus, atopic dermatitis, etc.), the cause of the disease may lead to the unmasking of tolerance to an abnormal keratinocyte clone that remained hidden in these lines. In addition to epithelial cells, other cells may be involved in the occurrence of acquired blaschkolinear dermatoses. In linear atrophoderma and linear fibromatosis, the histogenesis seems to involve hypothetic dermal clones. The extension of an acquired dermatosis on a preexisting linear nevoid disorder is an argument in favor of an early embryonic somatic mutation of a skin cell line.

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Linear fixed drug eruption

Cited by 42 publications

References 4 publications

Calcium Acetate-Induced Linear Fixed Drug Eruption

Calcium Acetate-Induced Linear Fixed Drug Eruption

What is a Nevus

Acquired Blaschkolinear dermatoses

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