Left ventricular thrombus sans overt cardiac pathology

Yadava, Om Prakash; Yadav, Sumit; Juneja, Saurabh; Chopra, Vijay; Passey, R. D.; Ghadiok, R

doi:10.1016/s0003-4975(03)00198-x

Cited by 18 publications

(12 citation statements)

References 4 publications

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“…In our patient, it is not clear whether the LV thrombi embolized or were dissolved by lysis. However, there was no clinical or laboratory evidence of embolization, and the large extent of lysis in both thrombi is in favor of clot resolution although silent embolization cannot be absolutely excluded 2,6–10 . The echogenic and more organized attachment of the clot to the LV apex also raises the possibility of a firmer attachment, less prone to detachment and embolization, however this is entirely speculative.…”

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confidence: 96%

Morphological Assessment of Left Ventricular Thrombus by Live Three‐Dimensional Transthoracic Echocardiography

et al. 2004

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We report the usefulness of live three-dimensional transthoracic echocardiography (3DTTE) in the morphological assessment of a left ventricular thrombus. Using live 3DTTE, the thrombus could be easily viewed end-on and from the sides. In addition, by cropping the 3D images sequentially in transverse (horizontal or short axis), longitudinal (vertical or long axis), frontal, and oblique planes, the degree and extent of lysis within the thrombus, which represents an integral part of the clot-resolution process, could be comprehensively assessed. The site of attachment of the thrombus in the left ventricular apex and its morphology could also be fully evaluated in three dimensions by live 3DTTE.

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confidence: 96%

Morphological Assessment of Left Ventricular Thrombus by Live Three‐Dimensional Transthoracic Echocardiography

et al. 2004

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“…Moreover, since left ventricular thrombosis is rare in the absence of cardiac disease, such a thrombus may be overlooked, particularly when it is small or less mobile. Conversely, when the thrombus is large and mobile, it may be indistinguishable from cardiac myxoma [14][15][16][17], and this diagnostic uncertainty may lead to surgery. In our present patient, the thrombus was distinguished from myxoma by its relatively small size and its resolution after treatment.…”

Section: Discussionmentioning

confidence: 99%

Simultaneous Thrombosis in a Normal Left Ventricle and Normal Carotid Artery in a Patient With a Stroke Secondary to Iron Deficiency Anemia

Nakamizo

2014

J Med Cases

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Iron deficiency anemia (IDA) is implicated as a cause of stroke, particularly in young patients without cardiovascular disease. In such patients, thrombi sometimes form in carotid arteries or the aorta. We report here a patient with a stroke secondary to IDA with thrombi in the normal left ventricle and normal carotid artery. The patient was a 45-year-old woman with severe IDA who developed cerebral infarction in the right middle cerebral artery. She had no other thrombophilia or cardiovascular diseases. Echocardiography showed a left ventricular thrombus without cardiac disease, and the carotid ultrasound showed a mobile thrombus attached to the right internal carotid artery without atherosclerosis. Antithrombotic therapy with iron supplementation removed both thrombi within 2 weeks. This is the first case of IDA with a ventricular thrombus in a normal heart. This case identifies a new site for thrombosis in IDA and shows that patients with IDA may present with simultaneous thrombosis at separate sites. IDA must therefore be recognized as a cause of stroke or systemic embolism, particularly in patients without detectable cardiovascular disease. In such patients, it is important to conduct careful investigations to detect thrombosis.

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“…According to a 1993 meta-analysis of 11 studies involving 856 patients with an anterior MI, the OR for an embolic event was 5.5 (95% CI 3.0 to 9.8) for those with an LV mural thrombus documented on echocardiography 14. Most embolic complications occur within 3 or 4 months after thrombus development.…”

Section: Discussionmentioning

confidence: 99%

Myocardial infarction complicated by left ventricular thrombus and fatal thromboembolism following abrupt cessation of dabigatran

et al. 2014

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SUMMARYNovel anticoagulants are increasingly utilised in lieu of warfarin to treat non-valvular atrial fibrillation. Their clinical use in other non-FDA approved settings is also increasing. We present a case in which a patient abruptly stopped taking dabigatran due to a small bowel obstruction and shortly thereafter suffered a myocardial infarction complicated by left ventricular thrombosis with fatal embolisation to the superior mesenteric artery. In this context, we discuss the possibility of a rebound phenomenon of hypercoagulability with abrupt cessation of novel anticoagulants. BACKGROUND

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Left ventricular thrombus sans overt cardiac pathology

Cited by 18 publications

References 4 publications

Morphological Assessment of Left Ventricular Thrombus by Live Three‐Dimensional Transthoracic Echocardiography

Morphological Assessment of Left Ventricular Thrombus by Live Three‐Dimensional Transthoracic Echocardiography

Simultaneous Thrombosis in a Normal Left Ventricle and Normal Carotid Artery in a Patient With a Stroke Secondary to Iron Deficiency Anemia

Myocardial infarction complicated by left ventricular thrombus and fatal thromboembolism following abrupt cessation of dabigatran

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