Incidence of Inflammatory Rheumatic Diseases in Finland

Isomäki, H; Raunio, J; Essen, R von; Hämeenkorpi, R

doi:10.3109/03009747809095652

Cited by 74 publications

(35 citation statements)

References 7 publications

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“…We concluded that reactive arthritis may sometimes erroneously be diagnosed as rheumatoid arthritis. The same suggestion has been presented earlier by Isomaki et al (24) and by Calin and Marks (6). However, it is not excluded that in some cases reactive arthritis becomes chronic and leads into true rheumatoid arthritis.…”

supporting

confidence: 79%

IgA yersinia antibodies in yersinia arthritis

Granfors

Leino

Vuento

et al. 1983

Scandinavian Journal of Rheumatology

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We have followed the appearance and persistence of yersinia antibodies of different Ig classes during and after an acute yersinia infection, and compared the antibody responses of patients either developing arthritis after yersinia infection or not. The arthritic patients had higher concentration of lgCi and IgA class antibodies in the acute phase of infection than those remaining free of joint symptoms. On the other hand, the concentration of IgM class antibodies was slightly lower in arthritic patients. In addition, IgG and IgA class antibodies persisted in arthritic patients even for many years. The level and the persistence of the IgA, but not the IgG, response correlated to HLA-B27. In all patients the IgA class antibodies are composed of both IgAl-and IgA2-class immunoglobulins. However, anti-yersinia secretory IgA was detected in higher concentrations in the sera of those who developed arthritis. This as well as the high proportional concentration of lgA2 antibodies to Yersinia in the sera of these patients suggest an altered immune response of the gut in yersinia arthritis. Scand J Rheumatol Downloaded from informahealthcare.com by University of Toronto on 11/04/14 For personal use only. Scand J Rheumatol 7. Ahlqvist J . A hypothesis on the pathogenesis of rheumatoid and other non-specific synovitides based on a combination of observations often overlooked. Med Hypotheses 9: I I , 1982. Ahlqvist J , Ahvonen P. Risanen J , Wallgren G. Enteric infection with Yersinia enterocolitica. Large pyroninophilic cell reaction in mesenteric lymph nodes associated with early product ion of specific antibodies. Acta Pathol Microbiol Scand (A) 79: 109, 1971. Ahvonen P. Human yersiniosis in Finland. 1. Bacteriology and serology. Ann Clin Res 4: 30, 1972. Andre C, Berthoux F C, Andre F, Gillon J, Genin C, Sabatier J C. Prevalence of lgA2 deposit in IgA nephropathies. A clue to their pathogenesis. N Engl J Med 303: 1343, 1980. Brandtzaeg P. The humoral immune system of the human gastro-intestinal tract. Monogr Allergy 17: 195. 1981. Calin A, Marks S H. The case against seronegative rheumatoid arthritis. Am J Med 70: 992, 1981. Childerstone M, Colthorpe D, Ebringer A, Rook G . Ebringer R. James D C 0. Antibodies to Klebsiella measured in ankylosing spondylitis patients using ELISA. 5th European Immunology Meeting, Istanbul 1982 (abstract p. 214). Scand J Rheumatol Downloaded from informahealthcare.com by University of Toronto on 11/04/14 For personal use only.

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supporting

confidence: 79%

IgA yersinia antibodies in yersinia arthritis

Granfors

Leino

Vuento

et al. 1983

Scandinavian Journal of Rheumatology

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“…Patients with ReA comprise about 10% of those presenting at early synovitis clinics (45,46). While no large analysis on the distribution of the background infections in patients with ReA has been undertaken, it seems evident that previously the most frequent triggering infections in hospital-based series were due to Yersinia and Chlamydia trachomatis (45,47).…”

Section: Hospital-based Series Of Reamentioning

confidence: 98%

Reactive arthritis

Leirisalo‐Repo

2005

Scandinavian Journal of Rheumatology

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Reactive arthritis (ReA) was known as Reiter's disease or Fiessinger-Leroy disease for nearly 100 years. However, during the past 30 years the disease has been known as reactive arthritis, a member of the spondyloarthritis family. Despite knowing the initiating event (infection) and genetic constitution (many patients have HLA-B27) of ReA, a model of interplay between environment and genetics, its pathogenesis is still incompletely known. This review covers the epidemiology, clinical features, treatment, and prognosis of ReA.

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“…A retrospective study in Heinola, Finland used several different strategies: hospital records, interviews of physicians, and official statistics, and reported the annual incidence of all arthritis in adults 218/100,000 (19). The incidence of definite rheumatoid arthritis in adults was 40.…”

Section: Discussionmentioning

confidence: 99%

Incidence of arthritis in urban finnish children: A prospective study

Kunnamo

Kallio²,

Pelkonen

1986

Arthritis & Rheumatism

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A Prospective Study ILKKA KUNNAMO, PENTTI KALLIO, and PIRKKO PELKONEN The incidence of various types of arthritis in children was estimated by a prospective l-year study in the greater Helsinki area (population under 16 years of age: 148,362). Patients were sought from primary care physicians, and 71% of the patients studied were seen within 1 week of the onset of symptoms. All patients recehed followup care for at least 3 months; patients whose symptoms were prolonged received followup care for a minimum of 2 years. The incidence per 100,000 children under 16 years of age was as follows: 108.5 for all cases of arthritis, 6.7 for septic arthritis, 5.4 for enteropathic arthritis, 51.9 for transient synovitis of the hip, 18.9 for prolonged arthritis (duration >3 months), and 25.8 for acute transient arthritis. The incidence of juvenile rheumatoid arthritis was 19.6. Oligoarticular disease was prevalent (76%) among the juvenile rheumatoid arthritis patients.Few reports have been published regarding the incidence (1-7) and spectrum (8) of arthritis in children. Incidence studies are almost always concerned with juvenile rheumatoid arthritis (JRA) (1-5) and acute rheumatic fever (6-7). Little is known of the frequency of other types of arthritis in children.

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Incidence of Inflammatory Rheumatic Diseases in Finland

Cited by 74 publications

References 7 publications

IgA yersinia antibodies in yersinia arthritis

IgA yersinia antibodies in yersinia arthritis

Reactive arthritis

Incidence of arthritis in urban finnish children: A prospective study

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