Incidence of Contrast-induced Nephropathy in Kidney Transplant Recipients

Haider, Mumnoon; Yessayan, Lenar; Venkat, K. K.; Goggins, M.; Patel, Anita; Karthikeyan, Vanji

doi:10.1016/j.transproceed.2015.01.008

Cited by 28 publications

(23 citation statements)

References 7 publications

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“…The patient presented a mild alteration of the renal function (serum creatinine: 1.3 mg/dL and estimated glomerular filtration rate: 68 mL/min), and therefore an endovascular approach could have been an excessive risk for the patient. For the same reason, no contrast-enhanced computed tomography angiography scan was performed, since the ultrasound duplex scan had been sufficient in achieving preoperative information [13, 14]. …”

Section: Discussionmentioning

confidence: 99%

Giant true Brachial Artery Aneurysm after Hemodialysis Fistula Closure in a Renal Transplant Patient

Ferrara

Filippo

Spalla

et al. 2016

Case Rep Nephrol Dial

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The usual manifestation of brachial artery aneurysms is the incidental finding of a swelling of the arm, combined with paresthesia or pain in some cases. The etiology is often traumatic or secondary to drug abuse. Pathophysiology of brachial artery dilation in these cases is not completely clear. We herein describe a case of a 61-year-old male presenting with a giant, painful, pulsatile mass on his left arm. He was submitted to a cadaveric kidney transplant in 2005. He had a functioning arteriovenous fistula (AVF) on his right arm, and a spontaneously thrombosed radiocephalic AVF on his left arm. The aneurysm was surgically resected, sparing the median nerve that was totally entrapped and an inverted segment of the basilic vein interposed. At the follow-up, the patient did not present neurological or ischemic disturbs, and the vein graft maintained its patency.

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Section: Discussionmentioning

confidence: 99%

Giant true Brachial Artery Aneurysm after Hemodialysis Fistula Closure in a Renal Transplant Patient

Ferrara

Filippo

Spalla

et al. 2016

Case Rep Nephrol Dial

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“…The main insult is the ensuing vasoconstriction of the afferent glomerular arterioles and reduction in renal blood flow and glomerular filtration rate. Renal vasoconstriction, as well as direct tubular epithelial toxicity, is the two major mechanisms by which contrast causes AKI as explained by Haider et al[9] in 2015. Immediately after contrast use, there is a transient increase in renal blood flow followed by a prolonged reduction in flow resulting in renal ischemia.…”

Section: Contrast Nephropathy In Renal Transplantationmentioning

confidence: 99%

“…Area under the curve (AUC) for age, FK506 levels, daily Cellcept dose, baseline creatinine, eGFR, and volume of IV contrast were 0.60, 0.64, 0.63, 0.57, 0.63, and 0.68, respectively[9]. Adapted from Haider et al[9], Incidence of Contrast-induced Nephropathy in Kidney Transplant Recipients. Transplantation Proceedings 2015; 47: 2379-2383 (with permission).…”

Section: Contrast Nephropathy In Renal Transplantationmentioning

confidence: 99%

“…To date, Haider et al[9] (2015) study is considered the largest retrospective study evaluating the incidence of CIN in kidney transplantation. The incidence of CIN in this study was low (5.6%), much lower than reported by Ahuja et al[10] (2000).…”

Section: Contrast Nephropathy In Renal Transplantationmentioning

confidence: 99%

“…The incidence of CIN in this study was low (5.6%), much lower than reported by Ahuja et al[10] (2000). Two fundamental factors were implicated in this low incidence of CIN in this landmark study first: the relatively elevated baseline eGFR (> 70 mL/min per 1.73 m 2 ) and second: The use of “hypo-osmolar” contrast applied in this procedure[9]. …”

Section: Contrast Nephropathy In Renal Transplantationmentioning

confidence: 99%

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“Contrast nephropathy” in renal transplantation: Is it real?

Abbas¹,

Julie²,

Sharma³

et al. 2016

WJT

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The risk of contrast-induced nephropathy (CIN) in renal transplant recipients is increased in diabetics, patients with impaired basal kidney function, patients in shock, patients presenting with acute emergency and in old age recipients. Approximately one-third of all hospitalized patients with acute kidney injury is attributed to CIN. In the United States, it is the third leading cause of hospital-acquired renal failure. Therefore, efforts should be directed to minimize CIN-related morbidity and mortality as well as to shorten hospital stay. While the role of peri-procedural prophylactic hydration with saline is unequivocal; the use of acetyl cysteine is not based on robust evidence. The utility of theophylline, aminophylline, calcium channel blockers, natriuretic peptide, and diuretics does not have proven role in attenuating CIN incidence. We aim to analyze the evidence for using various protocols in published literature to limit CIN-associated morbidity and mortality, particularly during surveillance of the renal allograft survival.

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What Are the Unique Considerations in Renal Transplant Patients with Abdominal Pain?

Potter

Schooler

2019

Gastrointestinal Emergencies

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Incidence of Contrast-induced Nephropathy in Kidney Transplant Recipients

Cited by 28 publications

References 7 publications

Giant true Brachial Artery Aneurysm after Hemodialysis Fistula Closure in a Renal Transplant Patient

Giant true Brachial Artery Aneurysm after Hemodialysis Fistula Closure in a Renal Transplant Patient

“Contrast nephropathy” in renal transplantation: Is it real?

What Are the Unique Considerations in Renal Transplant Patients with Abdominal Pain?

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