“…The level of coronary occlusion, presence of large collaterals from the LAD, myocardial hypertrophy and ventricular dilatation should also be taken into account when addressing the malperfusion syndrome [12]. The incidence acute malperfusion syndrome ranges from 0.3% to 1.9% [13,14]. Delayed hypoperfusion of IMA is usually due to graft thrombosis, progression of atherosclerosis, kinking secondary to surgical technique, underdevelopment due to competitive flow, vaso-steal due to untied side branches or anomalous IMA with a large caliber lateral internal thoracic artery [10,15 -18].…”