Hypoplastic Inferior Vena Cava with Aplasia of the Right Kidney and Absence of the Right Adrenal Gland

Provost, John M.; Hoffman, Carl M.; Ryan, John F.; Vines, Donald H.

doi:10.1097/00000658-197111000-00016

Cited by 11 publications

(6 citation statements)

References 9 publications

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“…Hypotheses are either embryonic dysontogenesis that affects separate segments or the entire IVC and intrauterine thrombosis or umbilical vein complication postpartum as causal events. There can be an association between anomalies of the IVC and congenital malformation of abdominal viscera such as polysplenia, asplenia, situs inversus and others [2–4]. Usually those patients are diagnosed early in life.…”

Section: Discussionmentioning

confidence: 99%

Recurrent deep venous thrombosis caused by congenital interruption of the inferior vena cava and heterozygous factor V Leiden mutation

Schneider¹,

Eynatten²,

Dugi³

et al. 2002

Journal of Internal Medicine

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Abstract. Schneider JG, Eynatten MV, Dugi KA, Duex M, Nawroth PP (Ruprecht-Karls-University of Heidelberg, Heidelberg, Germany). Recurrent deep venous thrombosis caused by congenital interruption of the inferior vena cava and heterozygous factor V Leiden mutation (Case report). J Intern Med 2002; 252: 276-280.A case of a 44-year-old patient with recurrent deep venous thrombosis (DVT) caused by congenital dysgenesis of the inferior vena cava (IVC) in coincidence with heterozygous factor V Leiden mutation is presented. The IVC malformation was a fortuitous finding because the vascular malformation of the collateral draining thoracic veins were suspected to be a malignant mass in chest X-ray. This vascular abnormality is a rare finding but recent epidemiological research suggests that there may be an association between the congenital absence of the IVC and DVT. In our case, the patient is even at higher risk combining the malformation probably affecting venous blood flow and the hypercoagulabilic 1 state by heterozygous presence of the factor V Leiden mutation.

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Section: Discussionmentioning

confidence: 99%

Recurrent deep venous thrombosis caused by congenital interruption of the inferior vena cava and heterozygous factor V Leiden mutation

Schneider¹,

Eynatten²,

Dugi³

et al. 2002

Journal of Internal Medicine

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“…Hemiazygos continuation is reported less frequently than azygos continuation 14 . The intermediate channels, the gonadal, perirenal and periureteric veins, are less often used 27 . The vertebrolumbar route is involved in 95% of cases and is the sole route in 70% of such cases 25 .…”

Section: Discussionmentioning

confidence: 99%

“…The vertebrolumbar route is involved in 95% of cases and is the sole route in 70% of such cases 25 . Anterior wall veins are only used in 15% of cases, 19,27−29 and in almost all of these, the vertebrolumbar route is also used 21 . Flow through the anterior abdominal wall veins requires much more pressure to overcome venous valves and is only used if the common iliac veins are thrombosed 21,26 .…”

Section: Discussionmentioning

confidence: 99%

“…There is an increasing body of adults found incidentally 8,17 , 38 with this group of harmless anomalies. In addition to venous insufficiency, 39 deep venous thrombosis 36,40 and dilated abdominal wall veins, 27,29 this anomaly has presented with bleeding, urinary retention and abdominal and thoracic pain.…”

Section: Discussionmentioning

confidence: 99%

“…The importance of recognizing this entity is primarily in avoiding unnecessary thoracotomies 41,43 and laparotomies 22,27 . It should be noted that, in the first clinical case reported, ligation of the azygos vein resulted in the patient's death 43 .…”

Section: Discussionmentioning

confidence: 99%

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Developmental anomaly of the inferior vena cava

Sandercoe

Brooke‐Cowden

2003

ANZ Journal of Surgery

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Despite the many possible modes of presentation, congenital anomalies of the inferior vena cava are increasingly being found in asymptomatic patients. Although plain chest radiography may reveal enough suggestive signs, the diagnosis is usually confirmed by ultrasound, computed tomography and magnetic resonance imaging. The authors present a case of infrarenal absence of the inferior vena cava that presented as a retroperitoneal mass and discuss the embryology and clinical implications of this unusual entity.

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