Hypertrophie des rechten Herzens aus der Sicht des Klinikers

Ulmer, W. T.

doi:10.1007/978-3-642-72305-6_9

Cited by 12 publications

(2 citation statements)

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“…[Harris et al, 1961]. Zum Teil zeigen die Arbeiten aber bei entsprechenden Sauerstoff druckwerten wesentlich höhere Drücke in der A. pulmonalis [Ulmer, 1972], Bei diesen Patienten ist aber zu diskutieren, inwieweit nicht ein direkter mechanischer Einfluss auf die pulmonale Hämodynamik durch den in grösseren Alveolargebieten herrschenden er höhten Alveolardruck, hervorgerufen durch die erhöhten Strömungswiderstände in den Atemwegen, besteht [Harris et al, 1968;Schott et al, 1979].…”

Section: Gruppe B Hypoxie Und Hyperkapnieunclassified

“…Hiermit wird auch die klinische Beob achtung, dass die Cor-pulmonale-Entwicklung so unterschiedlich stark ausfällt, eine Klärung finden, wenn auch die abnehmen de Hypertrophiefähigkeit des rechten Ven trikels sowie der sonstige Zustand des Gefässbettes der Lunge mit zunehmendem Alter sicher auch zusätzliche Faktoren sind, welche die Entwicklung des chroni schen Cor pulmonale beeinflussen [Ulmer, 1972].…”

Section: Gruppe C Stenoseatmungunclassified

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Experimentelle Untersuchungen über kardiopulmonale Relationen bei Hypoxie, Hyperkapnie und Atemwegsobstruktion

Kowalski¹,

Islam²,

Almeida³

et al. 1980

Respiration

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On 18 dogs the effects of hypoxia (12, 10, 7.5% O₂) and hypoxia and hypercapnia (10% O₂ + 5, 7.5, 11% CO₂) on breathing, pulmonary vascular resistance, cardiac output and on the arterial and venous oxygen pressure were investigated. The relationship between arterial oxygen pressure to Pap as to vascular resistance is not in the mean linear. There are good, bad and nearly nonresponders in respect to the vascular resistance on the dogs. Therefore, calculation and evaluation of mean values are less helpful. A similar variation on men could help to understand also better the large scatter which is always shown on correlations between PO₂a: Pap or PO₂a : vascular resistance. Also the different pattern of the V/Q relationship may be influenced by the amount of the vascular response to local alveolar hypoxia. An increase of PCO₂a potentiates the local vascoconstrictor effect of hypoxia. Mainly this was seen in the range between hypocapnia and moderate rates of hypercapnia. Hypoxia shows an increase of breathing frequency, hypercapnia shows an increase in tidal volume. Also under hypercapnia there are nonresponders and better responders in response to the vascular resistance in the lung circulation. Mechanical airway obstruction is followed by an increase of tidal volume; obstruction caused by acetyl-choline is followed by a decrease of tidal volume and an increase of breathing frequency. Changes in the arterial blood gases are caused by changes in the ventilation perfusion relationship only under acetylcholine obstruction. Influences on the lung circulation by the airway obstruction under these experimental conditions as direct effects are small or not existent.

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Section: Gruppe B Hypoxie Und Hyperkapnieunclassified

Section: Gruppe C Stenoseatmungunclassified