Hyperbilirubinaemia and Perceptive Deafness

Fisch, L.; Norman, Arne

doi:10.1136/bmj.2.5245.142

Cited by 18 publications

(6 citation statements)

References 5 publications

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“…The sensorineural hearing loss associated with haemolytic disease of the newborn can occur without simultaneous athetosis or other neurological damage (15). On the other hand, hearing loss associated with neonatal hyperbilirubinaemia in otherwise healthy, fullterm infants without iso-immunization has not, to our knowledge, been described previously with certainty (3,8,12,15,16,17,23).…”

Section: Sensorineural Hearing Defectsmentioning

confidence: 89%

“…In Sweden a similar figure has been reported by Barr & Klockhoff (2); among a series of children with athetosis they found that 70 % had neurogenic hearing damage. The development of sensorineural hearing loss has also been described in infants with haemolytic disease of the newborn due to A or B immunization (8,15), and in pLemature infants with hyperbilirubinaemia without iso-immunization (8,16). The sensorineural hearing loss associated with haemolytic disease of the newborn can occur without simultaneous athetosis or other neurological damage (15).…”

Section: Sensorineural Hearing Defectsmentioning

confidence: 97%

“…The cause of the sensorineural hearing loss in haemolytic disease of the newborn is believed to be deposition of bilirubin in the cochlear nuclei (8,16), but damage to the cochlea has also been considered a possibility (9). The hearing loss mainly affects the higher frequencies, with a maximum degree of impairment in the range of 2 000 to 4 000 c/s (8,16). As a rule the hearing loss is approximately equal on both sides (16), but unilateral deafness has also been described (15).…”

Section: Sensorineural Hearing Defectsmentioning

confidence: 99%

“…It is well documented that damage to the central nervous system can occur in the newborn as a result of hyperbilirubinaemia in association with haemolytic disease and prematurity. The level of hyperbilirubinaemia that can be tolerated in the healthy, full-term infant without iso-immunization is, however, still uncertain (3,4,6,8,11,12,13,17,18,23,24,25,27). The aim of this investigation was to elucidate this question by a detailed follow-up of 226 children, half of whom had had neonatal hyperbilirubinaemia with a maximal bilirubin value exceeding 20 mg/100 ml and the other half of whom had had no clinical jaundice.…”

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confidence: 99%

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A Follow‐up Study of Hyperbilirubinaemia in Healthy, Full‐term Infants Without Iso‐immunization

Bengtsson

Verneholt

1974

Acta Paediatrica

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Summary With the aim of shedding light on the indications for exchange transfusion in healthy, full term infants without iso‐immunization, a detailed follow‐up investigation was made of 226 children, of whom 111 had had neonatal hyperbilirubinaemia with maxima) bilirubin values of 20–51 mg/100 ml and the other 115 (controls) had had no visible jaundice neonatally. The children underwent physical and neurological examinations as well as audiometric and psychological tests. Among the 111 children with neonatal hyperbilirubinaemia 2 had coordination disturbances of the athetotic type and 4 had sensorineural hearing impairment, while none of the 115 control children had such disorders. The differences in the frequencies of the e conditions were not statistically significant, however. Neither did the frequencies of subnormal intelligence and psychological disturbances in the two groups differ significantly. Exchange transfusion is recommended in healthy, full‐term infants without iso‐immunization as'a measure to prevent the serum bilirubin level from exceeding 25 mg 100 ml.

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Section: Sensorineural Hearing Defectsmentioning

confidence: 89%

Section: Sensorineural Hearing Defectsmentioning

confidence: 97%

Section: Sensorineural Hearing Defectsmentioning

confidence: 99%

mentioning

confidence: 99%

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A Follow‐up Study of Hyperbilirubinaemia in Healthy, Full‐term Infants Without Iso‐immunization

Bengtsson

Verneholt

1974

Acta Paediatrica

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“…The severity of hearing loss could not be predicted from the severity of the athetosis. Fisch and Norman (1961) assessed the hearing of 50 children jaundiced at birth and found six with partial deafness. Padden (1959) tested the ability of 105 deaf and hard-of-hearing male college students to orient themselves when submerged in water with eyes open and blindfolded.…”

Section: Multiple Handicapsmentioning

confidence: 99%

Chapter IV: The Deaf and the Hard of Hearing

Lane¹

1963

Review of Educational Research

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INVESTIGATORS in problems of the deaf and the hard of hearing have collected their research, organized their results, and published books that are valuable to the students preparing to teach, to test, and to continue research.Two of these were published at the same time and under the same title, but here the similarity ceased. Levine (1960) wrote her text from the rehabilitation point of view, stressed the psychosocial problems of deafness, and thus provided a guide for counselors and students interested in clinical testing. Myklebust (1960) stressed the limitations produced by sensory deprivation on the balance and equilibrium of all psychological processes. He included the results of his experimental work and many of the previously unpublished studies of his graduate students. Davis and Silverman (1960) revised Hearing and Deafness into a textbook for beginning students of audiology. They kept the original chapters intact, with additions to bring them up to date, and replaced the chapter on military aural rehabilitation with a new chapter on vocational guidance. Canfield (1959) wrote his handbook for the use of the person with a hearing impairment and his family.Wever translated and arranged the experiments of Von Bekesy (1960) into a volume that presents problems of auditory research, critical experiments in the field of hearing, and a discussion of cochlear mechanics.

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Neonatal Bilirubinemia in Production of Long-Term Neurological Deficits

Holmes¹

1968

Arch Pediatr Adolesc Med

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ASSOCIATION of marked bilirubinemia in the early neonatal period with kernicterus is well established. The possibility has occurred to many that lesser degrees of bilirubinemia may be related to less severe cerebral injury, too mild to be detected during the newborn period, but recognizable in later years as mild to moderate impairment of intelligence, partial nerve deafness, and other neurological deficits. A closely analogous concept is that of a "continuum of reproductive casualty" developed by Pasamanick and Knobloch.1 It was the purpose of the present study to investigate the application of such a hypothesis to children with mild to moderate neonatal bilirubinemia.In preparation for this study, the world literature was reviewed. The most significant reports are summarized in Table 1. This illustrates the conflicting results and complexities of relating moderate bilirubinemia to subsequent cerebral difficulties in surviving children. Examination of Table 1 reveals that a number of factors were not always taken into account. The maturity of the infants was not always stated. There was no control group in seven of the ten studies. The levels of serum bilirubin varied considerably and were not always stated precisely. The in¬ cidence of exchange transfusions varied from none to some or was not stated. The follow-up examinations were carried out on children over a wide age range. Many of them were only 3 to 4 years old or

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Hyperbilirubinaemia and Perceptive Deafness

Cited by 18 publications

References 5 publications

A Follow‐up Study of Hyperbilirubinaemia in Healthy, Full‐term Infants Without Iso‐immunization

A Follow‐up Study of Hyperbilirubinaemia in Healthy, Full‐term Infants Without Iso‐immunization

Chapter IV: The Deaf and the Hard of Hearing

Neonatal Bilirubinemia in Production of Long-Term Neurological Deficits

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