Human cysticercosis: Parasitology, pathology, clinical manifestations and available treatment

Webbe, G.

doi:10.1016/0163-7258(94)90038-8

Cited by 32 publications

(18 citation statements)

References 84 publications

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“…The small number of cysts is this study is in agreement with the profile found in most NC patients (36,37). Two studies also performed in Latin America have indicated a small number of cysts per patient, one showing a range from 1.9 to 5.0 cysts, and the other a mean of 3.7 cysts (38,39).…”

Section: Discussionsupporting

confidence: 76%

Serum and cerebrospinal fluid S100B concentrations in patients with neurocysticercosis

Lima

Walz

Tort

et al. 2006

Braz J Med Biol Res

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The clinical manifestations of neurocysticercosis (NC) are varied and depend on the number and location of cysts, as well as on the host immune response. Symptoms usually occur in NC when cysticerci enter a degenerative course associated with an inflammatory response. The expression of brain damage markers may be expected to increase during this phase. S100B is a calcium-binding protein produced and released predominantly by astrocytes that has been used as a marker of reactive gliosis and astrocytic death in many pathological conditions. The aim of the present study was to investigate the levels of S100B in patients in different phases of NC evolution. Cerebrospinal fluid and serum S100B concentrations were measured in 25 patients with NC: 14 patients with degenerative cysts (D), 8 patients with viable cysts (V) and 3 patients with inactive cysts. All NC patients, except 1, had five or less cysts. In most of them, symptoms had been present for at least 1 month before sample collection. Samples from 8 normal controls (C) were also assayed. The albumin quotient was used to estimate the blood-brain barrier permeability. There were no significant differences in serum (P = 0.5) or cerebrospinal fluid (P = 0.91) S100B levels among the V, D, and C groups. These findings suggest that parenchymal changes associated with a relatively small number of degenerating cysts probably have a negligible impact on glial tissue.

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Section: Discussionsupporting

confidence: 76%

Serum and cerebrospinal fluid S100B concentrations in patients with neurocysticercosis

Lima

Walz

Tort

et al. 2006

Braz J Med Biol Res

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“…This patient (D12 on Table 2) had at least 30 cysts (most of them viable forms) and also had normal NSE levels. Data from literature have indicated that the preponderance of cases with NC present 10 or less cysts [25]. In a review of epidemiological studies performed in Brazil, 67% of NC patients had intraparenchymal parasites.…”

Section: Discussionmentioning

confidence: 99%

Neuron-specific enolase in patients with neurocysticercosis

Lima

Takayanagui

Garcia

et al. 2004

Journal of the Neurological Sciences

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“…As it was described in literature, NC tends to take from 23 months to 7 years to calcify [7], it was demonstrated that in our sample the process was an accelerated one. In spite of the fact that treatment with anticyrcotic drugs does not extinguish the parasite, completely, from the CNS, in many cases, it can accelerate the calcification process [8][9][10][11][12][13][14][15][16][17][18][19][20]. As the CT findings at stage IV were statistically significant in relation to the normal neurological exam, we believe that the patients have been benefited by the treatment.…”

Section: Discussionmentioning

confidence: 99%

Clinical tomographic correlations of 220 patients with neurocisticercosis, Bahia, Brazil

Andrade-Filho¹,

Figuerôa²,

Andrade-Souza³

2007

Braz J Infect Dis

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Neurocysticercosis (NCC) is a common parasitic disease in our region, presenting diversity of neurological symptoms and signs. The present study has as primary objective an evaluation of the NCC's clinical and epidemiological profile within Bahia State, by means of a prospective study of 220 patients diagnosed from March 1988 to March 1999, with a follow-up of six months. Exams, such as Computed Cranial Tomography Scan (CT), Cerebral Spine Fluid (CSF) and Electroencephalogram (EEG), were accomplished in three distinct moments of these patients' evolution: at starting or diagnostic point (zero time), at after-intervention period (one month after treatment), and at control period (six months after treatment).

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Human cysticercosis: Parasitology, pathology, clinical manifestations and available treatment

Cited by 32 publications

References 84 publications

Serum and cerebrospinal fluid S100B concentrations in patients with neurocysticercosis

Serum and cerebrospinal fluid S100B concentrations in patients with neurocysticercosis

Neuron-specific enolase in patients with neurocysticercosis

Clinical tomographic correlations of 220 patients with neurocisticercosis, Bahia, Brazil

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