Homocysteine and related B-vitamin status in coeliac disease: Effects of gluten exclusion and histological recovery

Abstract

Gluten exclusion in CD improves folate status and normalizes homocysteine concentrations. Reducing the risk of homocysteine-related disease may be another reason for aggressive diagnosis and treatment of CD.

Hypercoagulability in celiac disease — An update

“…Nutritional deficiencies B12, folate, protein C and S deficiency due to vitamin K deficiency [64,66] Genetic diseases MTHFR variants [39] Thrombophylic autoantibodies Antiphospholipid, antiprothrombin, antiphosfatidylserine/ prothrombin [17,22,26,27,45,47,52,53,55,66] Amino acid abnormalities Hyperhomocystinemia [41,48,[60][61][62][63]66] Autoimmune homology Mimicry between tTG/ factor XIII [67] Atherosclerosis Endothelial dysfunction [22,24] …”

Nutrition et fausses couches spontanées : une revue de la littérature

“…L'hyperhomocysté iné mie identifié e au cours de la MC est é galement impliqué e [119]. Les patientes atteintes de MC non traité es pré sentent en effet des taux d'homocysté ine significativement plus é levé s et des taux de folates significativement plus bas.…”

Hematologic manifestations of celiac disease

“…There was no relationship between low B12 levels and clinical characteristics. 55 The cause of vitamin B12 deficiency in CD is unclear. It may include decreased gastric acid, bacterial overgrowth, autoimmune gastritis, decreased efficiency of mixing with transfer factors in the intestine, or perhaps dysfunction (inflammation) of the distal small intestine.…”